(Q43106561)

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High glucose activates HIF-1-mediated signal transduction in glomerular mesangial cells through a carbohydrate response element binding protein

scientific article published on 7 April 2010

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scholarly article

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Europe PubMed Central

PubMed publication ID

14 November 2017

High glucose activates HIF-1-mediated signal transduction in glomerular mesangial cells through a carbohydrate response element binding protein (English)

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Europe PubMed Central

PubMed publication ID

14 November 2017

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Tsubasa Isoe

1

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14 November 2017

Yuichi Makino

2

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14 November 2017

Katsutoshi Mizumoto

3

1 reference

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14 November 2017

Hidemitsu Sakagami

4

1 reference

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14 November 2017

Yukihiro Fujita

5

1 reference

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14 November 2017

Jun Honjo

6

1 reference

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14 November 2017

Yumi Takiyama

7

1 reference

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14 November 2017

Hiroshi Itoh

8

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14 November 2017

Masakazu Haneda

9

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14 November 2017

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publication date

7 April 2010

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14 November 2017

Kidney International

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14 November 2017

78

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14 November 2017

1

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14 November 2017

48-59

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Europe PubMed Central

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14 November 2017

https://scigraph.springernature.com/pub.10.1038/ki.2010.99

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The pathogenesis of diabetic nephropathy

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The effect of intensive treatment of diabetes on the development and progression of long-term complications in insulin-dependent diabetes mellitus. The Diabetes Control and Complications Trial Research Group

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Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33). UK Prospective Diabetes Study (UKPDS) Group

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GLUT-1 overexpression: Link between hemodynamic and metabolic factors in glomerular injury?

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Genetics of Kidneys in Diabetes (GoKinD) study: a genetics collection available for identifying genetic susceptibility factors for diabetic nephropathy in type 1 diabetes

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Role of advanced glycation end products in diabetic nephropathy.

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Reactive oxygen species from mitochondria induce cyclooxygenase-2 gene expression in human mesangial cells: potential role in diabetic nephropathy

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Cellular mechanisms in the development and progression of diabetic nephropathy: activation of the DAG-PKC-ERK pathway.

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Suppression subtractive hybridization identifies high glucose levels as a stimulus for expression of connective tissue growth factor and other genes in human mesangial cells

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Overview of Glucose Signaling in Mesangial Cells in Diabetic Nephropathy

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Plasminogen activator inhibitor-1 and diabetic nephropathy.

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Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

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Dimerization, DNA binding, and transactivation properties of hypoxia-inducible factor 1

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Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

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Hypoxic induction of Ctgf is directly mediated by Hif-1.

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Hypoxia induces hexokinase II gene expression in human lung cell line A549.

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Selection and analysis of a mutant cell line defective in the hypoxia-inducible factor-1 alpha-subunit (HIF-1alpha). Characterization of hif-1alpha-dependent and -independent hypoxia-inducible gene expression

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Induction of the Plasminogen Activator Inhibitor-1 Gene Expression by Mild Hypoxia Via a Hypoxia Response Element Binding the Hypoxia-Inducible Factor-1 in Rat Hepatocytes

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Oxygen sensing by HIF hydroxylases

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C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

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Signal transduction in hypoxic cells: inducible nuclear translocation and recruitment of the CBP/p300 coactivator by the hypoxia-inducible factor-1alpha

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Transforming growth factor beta1 induces hypoxia-inducible factor-1 stabilization through selective inhibition of PHD2 expression.

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HIF overexpression correlates with biallelic loss of fumarate hydratase in renal cancer: novel role of fumarate in regulation of HIF stability

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A non-hypoxic, ROS-sensitive pathway mediates TNF-alpha-dependent regulation of HIF-1alpha

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Induction of hypoxia-inducible factor-1alpha by transcriptional and translational mechanisms

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Altered Gene Expression Related to Glomerulogenesis and Podocyte Structure in Early Diabetic Nephropathy of db/db Mice and Its Restoration by Pioglitazone

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New insights into the function of the Wilms tumor suppressor gene WT1 in podocytes

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TNF-alpha, but not IFN-gamma, regulates CCN2 (CTGF), collagen type I, and proliferation in mesangial cells: possible roles in the progression of renal fibrosis

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Regulation of the hypoxia-inducible transcription factor 1alpha by the ubiquitin-proteasome pathway

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Identification of a functional hypoxia-responsive element that regulates the expression of the egl nine homologue 3 (egln3/phd3) gene

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Hypoxia-induced assembly of prolyl hydroxylase PHD3 into complexes: implications for its activity and susceptibility for degradation by the E3 ligase Siah2

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The human hypoxia-inducible factor 1alpha gene: HIF1A structure and evolutionary conservation

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Carbohydrate response element binding protein, ChREBP, a transcription factor coupling hepatic glucose utilization and lipid synthesis

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Up-regulation of HIF in experimental acute renal failure: evidence for a protective transcriptional response to hypoxia

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Increased glucose uptake and metabolism in mesangial cells overexpressing glucose transporter 1 increases interleukin-6 and vascular endothelial growth factor production: role of AP-1 and HIF-1alpha.

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Hyperglycemia regulates hypoxia-inducible factor-1alpha protein stability and function

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Transcriptional up-regulation of inhibitory PAS domain protein gene expression by hypoxia-inducible factor 1 (HIF-1): a negative feedback regulatory circuit in HIF-1-mediated signaling in hypoxic cells.

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Identifiers

10.1038/KI.2010.99

1 reference

Europe PubMed Central

PubMed publication ID

14 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 November 2017

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