(Q43234292)

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Donor IL-4-treatment induces alternatively activated liver macrophages and IDO-expressing NK cells and promotes rat liver allograft acceptance.

scientific article published on 26 November 2009

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12 February 2020

Donor IL-4-treatment induces alternatively activated liver macrophages and IDO-expressing NK cells and promotes rat liver allograft acceptance (English)

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12 February 2020

Suzanne J. Hodgkinson

4

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12 February 2020

6

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12 February 2020

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Chuanmin Wang

1

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12 February 2020

Szun Szun Tay

2

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12 February 2020

Giang T Tran

3

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12 February 2020

Richard D M Allen

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12 February 2020

Geoffrey W McCaughan

7

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12 February 2020

Alexandra F Sharland

8

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12 February 2020

G Alex Bishop

9

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12 February 2020

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26 November 2009

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12 February 2020

Transplant Immunology

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12 February 2020

22

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12 February 2020

3-4

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12 February 2020

172-178

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12 February 2020

The IL-4 receptor: signaling mechanisms and biologic functions

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRIM.2009.11.005

7 January 2021

Alternative activation of macrophages

1 reference

https://api.crossref.org/works/10.1016%2FJ.TRIM.2009.11.005

7 January 2021

Comparative roles of IL-4, IL-13, and IL-4Ralpha in dendritic cell maturation and CD4+ Th2 cell function.

1 reference

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7 January 2021

IL-4 induces in vivo production of IFN-gamma by NK and NKT cells

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Interleukin-4 induces human hepatocyte apoptosis through a Fas-independent pathway.

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Role of IL-4 and Th2 responses in allograft rejection and tolerance

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7 January 2021

The role of interleukin-4 in the induction phase of allogeneic neonatal tolerance

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7 January 2021

Are Th2 helper T lymphocytes beneficial, deleterious, or irrelevant in promoting allograft survival?

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Treatment with interleukin-4 prolongs allogeneic neonatal heart graft survival by inducing T helper 2 responses

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7 January 2021

Murine interleukin 4 transgenic heart allograft survival prolonged with down-regulation of the Th1 cytokine mRNA in grafts.

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Critical roles for IL-4, IL-5, and eosinophils in chronic skin allograft rejection

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Transfusion of polarized TH2-like cell populations into SCID mouse cardiac allograft recipients results in acute allograft rejection

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7 January 2021

Posttransplant interleukin-4 treatment converts rat liver allograft tolerance to rejection.

1 reference

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7 January 2021

Orthotopic liver allografts in the rat. The influence of strain combination on the fate of the graft

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7 January 2021

The essential roles of parenchymal tissues and passenger leukocytes in the tolerance induced by liver grafting in rats

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7 January 2021

Deletion of spontaneous rat liver allograft acceptance by donor irradiation

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7 January 2021

Paradoxical early immune activation during acceptance of liver allografts compared with rejection of skin grafts in a rat model of transplantation

1 reference

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7 January 2021

Crucial role of IL-4/STAT6 in T cell-mediated hepatitis: up-regulating eotaxins and IL-5 and recruiting leukocytes.

1 reference

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7 January 2021

Lethal hepatitis after gene transfer of IL-4 in the liver is independent of immune responses and dependent on apoptosis of hepatocytes: a rodent model of IL-4-induced hepatitis

1 reference

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7 January 2021

The role of indoleamine 2,3-dioxygenase in transplantation

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7 January 2021

Reversal of experimental allergic encephalomyelitis with non-mitogenic, non-depleting anti-CD3 mAb therapy with a preferential effect on T(h)1 cells that is augmented by IL-4

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7 January 2021

Evidence that apoptosis of activated T cells occurs in spontaneous tolerance of liver allografts and is blocked by manipulations which break tolerance

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7 January 2021

Heart allograft acceptance induced by anti-CD3 antibody in high-responder rats: effect on foxp3 and cytokine expression and graft infiltration

1 reference

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7 January 2021

Postoperative administration of donor B cells induces rat kidney allograft acceptance: lack of association with Th2 cytokine expression in long-term accepted grafts

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7 January 2021

A short course of cyclosporine immunosuppression inhibits rejection but not tolerance of rat liver allografts

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7 January 2021

Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair

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Ex vivo programmed macrophages ameliorate experimental chronic inflammatory renal disease

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Differential regulation of nitric oxide synthase-2 and arginase-1 by type 1/type 2 cytokines in vivo: granulomatous pathology is shaped by the pattern of L-arginine metabolism

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Relative contribution of direct and indirect allorecognition in developing tolerance after liver transplantation

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Tryptophan catabolism and T-cell tolerance: immunosuppression by starvation?

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Inhibition of allogeneic T cell proliferation by indoleamine 2,3-dioxygenase-expressing dendritic cells: mediation of suppression by tryptophan metabolites

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Function of indoleamine 2,3-dioxygenase in corneal allograft rejection and prolongation of allograft survival by over-expression

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Sleeping Beauty-based gene therapy with indoleamine 2,3-dioxygenase inhibits lung allograft fibrosis

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Blocking indoleamine dioxygenase activity early after rat liver transplantation prevents long-term survival but does not cause acute rejection.

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Overexpression of indoleamine dioxygenase in rat liver allografts using a high-efficiency adeno-associated virus vector does not prevent acute rejection.

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7 January 2021

Natural killer T cells from interleukin-4-deficient mice are defective in early interferon-? production in response to ?-galactosylceramide

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Indoleamine 2,3-dioxygenase is necessary for cytolytic activity of natural killer cells

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Inhibition of T cell proliferation by macrophage tryptophan catabolism

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NK cells promote islet allograft tolerance via a perforin-dependent mechanism

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NK cells promote transplant tolerance by killing donor antigen-presenting cells

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Posttransplant administration of donor leukocytes induces long-term acceptance of kidney or liver transplants by an activation-associated immune mechanism

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Early passenger leukocyte migration and acute immune reactions in the rat recipient spleen during liver engraftment: with particular emphasis on donor major histocompatibility complex class II+ cells

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