(Q43569912)

English

Failure to sustain recovery of Na,K-ATPase function is a possible mechanism for striatal neurodegeneration in hypoxic-ischemic newborn piglets

scientific article published in March 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Failure to sustain recovery of Na,K-ATPase function is a possible mechanism for striatal neurodegeneration in hypoxic-ischemic newborn piglets (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

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1 reference

based on heuristic

inferred from title

neurodegeneration

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based on heuristic

inferred from title

author name string

Golden WC

1

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Brambrink AM

2

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Traystman RJ

3

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Martin LJ

4

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

publication date

1 March 2001

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Europe PubMed Central

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24 November 2017

Molecular Brain Research

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Europe PubMed Central

PubMed publication ID

24 November 2017

88

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Europe PubMed Central

PubMed publication ID

24 November 2017

1-2

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Europe PubMed Central

PubMed publication ID

24 November 2017

94-102

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage

1 reference

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Excitotoxic cell death

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Peroxynitrite-mediated tyrosine nitration catalyzed by superoxide dismutase

1 reference

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Focal cerebral ischaemia induces a decrease in activity and a shift in ouabain affinity of Na+, K+-ATPase isoforms without modifications in mRNA and protein expression

1 reference

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7 January 2021

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Hypoxia-ischemia causes abnormalities in glutamate transporters and death of astroglia and neurons in newborn striatum.

1 reference

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Primary sensory and forebrain motor systems in the newborn brain are preferentially damaged by hypoxia-ischemia

1 reference

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Neuronal death in newborn striatum after hypoxia-ischemia is necrosis and evolves with oxidative stress

1 reference

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Chronic infusion ofl-glutamate causes neurotoxicity in rat striatum

1 reference

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Immunofluorescent localization of three Na,K-ATPase isozymes in the rat central nervous system: both neurons and glia can express more than one Na,K-ATPase

1 reference

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7 January 2021

based on heuristic

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Na+,K+-ATPase in developing fetal guinea pig brain and the effect of maternal hypoxia

1 reference

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7 January 2021

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Developmental expression of copper, zinc-superoxide dismutase in human brain by chemiluminescence

1 reference

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Isoforms of Na,K-ATPase alpha and beta subunits in the rat cerebellum and in granule cell cultures.

1 reference

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7 January 2021

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Selective sensitivity of synaptosomal membrane function to cerebral cortical hypoxia in newborn piglets

1 reference

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Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation

1 reference

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Inhibitory effect of several nitric oxide-generating compounds on purified Na+,K(+)-ATPase activity from porcine cerebral cortex.

1 reference

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7 January 2021

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Molecular cloning of three distinct forms of the Na+,K+-ATPase alpha-subunit from rat brain

1 reference

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7 January 2021

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Oxidant stress responses in premature infants during exposure to hyperoxia

1 reference

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Interventions for perinatal hypoxic-ischemic encephalopathy

1 reference

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Identifiers

10.1016/S0169-328X(01)00032-8

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 November 2017

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