(Q43610521)

25 November 2017

title

Neutrophils are primary source of O2 radicals during reperfusion after prolonged myocardial ischemia (English)

1 reference

25 November 2017

Duilio C

1

1 reference

25 November 2017

Ambrosio G

2

1 reference

25 November 2017

Kuppusamy P

3

1 reference

25 November 2017

DiPaula A

4

1 reference

25 November 2017

Becker LC

5

1 reference

25 November 2017

Zweier JL

6

1 reference

25 November 2017

publication date

1 June 2001

1 reference

American Journal of Physiology Heart and Circulatory Physiology

25 November 2017

published in

1 reference

25 November 2017

volume

280

1 reference

25 November 2017

issue

6

1 reference

25 November 2017

page(s)

H2649-57

1 reference

The relationship between oxygen radical generation and impairment of myocardial energy metabolism following post-ischemic reperfusion,

25 November 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

An anti-CD18 antibody limits infarct size and preserves left ventricular function in dogs with ischemia and 48-hour reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Identification of free radicals in myocardial ischemia/reperfusion by spin trapping with nitrone DMPO.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Direct evidence that oxygen-derived free radicals contribute to postischemic myocardial dysfunction in the intact dog.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Neutrophil accumulation in ischemic canine myocardium. Insights into time course, distribution, and mechanism of localization during early reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Neutrophil adherence to isolated adult canine myocytes. Evidence for a CD18-dependent mechanism.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Myocardial neutrophil accumulation during reperfusion after reversible or irreversible ischemic injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Deleterious effects of oxygen radicals in ischemia/reperfusion. Resolved and unresolved issues

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Antibody to CD-18 exerts endothelial and cardiac protective effects in myocardial ischemia and reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Postischemic free radical production in the venous blood of the regionally ischemic swine heart. Effect of deferoxamine

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Studies on the inhibitory mechanism of iodonium compounds with special reference to neutrophil NADPH oxidase

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Detection of hydroxyl radicals in the post-ischemic reperfused heart using salicylate as a trapping agent

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Adjunctive selectin blockade successfully reduces infarct size beyond thrombolysis in the electrolytic canine coronary artery model

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody (anti-Mo1, anti-CD11b) that inhibits leukocyte adhesion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Use of aromatic hydroxylation of phenylalanine to measure production of hydroxyl radicals after myocardial ischemia in vivo. Direct evidence for a pathogenetic role of the hydroxyl radical in myocardial stunning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Direct measurement of free radical generation following reperfusion of ischemic myocardium

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

Measurement of endothelial cell free radical generation: evidence for a central mechanism of free radical injury in postischemic tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.280.6.H2649

10.1152/AJPHEART.2001.280.6.H2649

21 January 2018

Identifiers

DOI

0 references

1 reference