(Q43660076)

English

B-T lymphocyte interactions in experimental autoimmune myasthenia gravis: antigen presentation by rat/mouse hybridoma lines secreting monoclonal antibodies against the nicotinic acetylcholine receptor

scientific article published in February 1988

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Europe PubMed Central

PubMed publication ID

25 November 2017

B-T lymphocyte interactions in experimental autoimmune myasthenia gravis: antigen presentation by rat/mouse hybridoma lines secreting monoclonal antibodies against the nicotinic acetylcholine receptor (English)

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

myasthenia gravis

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author name string

Zhang YP

1

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Tzartos SJ

2

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Wekerle H

3

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

language of work or name

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publication date

1 February 1988

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

European Journal of Immunology

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Europe PubMed Central

PubMed publication ID

25 November 2017

18

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Europe PubMed Central

PubMed publication ID

25 November 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

211-218

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Myasthenia gravis: stimulation of antireceptor autoantibodies by autoreactive T cell lines

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Intact B-cell activity is essential for complete expression of experimental allergic encephalomyelitis in Lewis rats.

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Molecular Forms and Hydrodynamic Properties of Acetylcholine Receptor from Electric Tissue

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Characteristics of monoclonal antibodies to denatured Torpedo and to native calf acetylcholine receptors: species, subunit and region specificity

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Demonstration of a main immunogenic region on acetylcholine receptors from human muscle using monoclonal antibodies to human receptor

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The rapid isolation of clonable antigen-specific T lymphocyte lines capable of mediating autoimmune encephalomyelitis

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Major histocompatibility complex-restricted antigen presentation to antigen-reactive T cells by B lymphocyte tumor cells

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Role of membrane immunoglobulin (Ig) crosslinking in membrane Ig-mediated, major histocompatibility-restricted T cell-B cell cooperation

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Antigen presentation is a function of all B cell subpopulations separated on the basis of size

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Interleukin-induced increase in Ia expression by normal mouse B cells

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I region-restricted antigen presentation by B cell–B lymphoma hybridomas

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Antigen presentation by Ia+ B cell hybridomas to H-2-restricted T cell hybridomas

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Flow Cytometry Analysis of Murine B Cell Lymphoma Differentiation

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Antigen-specific adherence of antibody-producing hybridoma cells: application to neural and nonneural antigens

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Transmembrane topography of nicotinic acetylcholine receptor: immunochemical tests contradict theoretical predictions based on hydrophobicity profiles

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Human T-helper lymphocytes in myasthenia gravis recognize the nicotinic receptor alpha subunit

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Regulation of T-cell function by antibodies: enhancement of the response of human T-cell clones to hepatitis B surface antigen by antigen-specific monoclonal antibodies

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Efficiency of antigen presentation to T cell clones by (B cell × B cell lymphoma) hybridomas correlates quantitatively with cell surface Ia antigen expression

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Ia antigen-bearing B cell tumor lines can present protein antigen and alloantigen in a major histocompatibility complex-restricted fashion to antigen-reactive T cells

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Ia-transfected L-cell fibroblasts present a lysozyme peptide but not the native protein to lysozyme-specific T cells

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The relation between major histocompatibility complex (MHC) restriction and the capacity of Ia to bind immunogenic peptides

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Decrease in macrophage antigen catabolism caused by ammonia and chloroquine is associated with inhibition of antigen presentation to T cells

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Functional significance of the regulation of macrophage Ia expression

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Identifiers

10.1002/EJI.1830180205

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

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