(Q43678421)

English

Drastic neuronal loss in vivo by beta-amyloid racemized at Ser(26) residue: conversion of non-toxic [D-Ser(26)]beta-amyloid 1-40 to toxic and proteinase-resistant fragments

scientific article published in January 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Drastic neuronal loss in vivo by beta-amyloid racemized at Ser(26) residue: conversion of non-toxic [D-Ser(26)]beta-amyloid 1-40 to toxic and proteinase-resistant fragments (English)

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

author name string

Kaneko I

1

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Morimoto K

2

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Kubo T

3

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

language of work or name

0 references

publication date

1 January 2001

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

104

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

1003-1011

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

Hydrogen peroxide mediates amyloid beta protein toxicity

1 reference

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Familial Alzheimer's disease-linked presenilin 1 variants elevate Abeta1-42/1-40 ratio in vitro and in vivo

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Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation.

1 reference

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Quantitative histological studies on the aged human brain. II. Senile plaques and neurofibrillary tangles in senile dementia (with an appendix on their occurrence in cases of carcinoma).

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Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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Computed circular dichroism spectra for the evaluation of protein conformation

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Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

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Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition

1 reference

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Thioflavine T interaction with synthetic Alzheimer's disease beta-amyloid peptides: detection of amyloid aggregation in solution

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Degradation of Alzheimer's beta-amyloid protein by human and rat brain peptidases: involvement of insulin-degrading enzyme.

1 reference

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Co-injection of beta-amyloid with ibotenic acid induces synergistic loss of rat hippocampal neurons.

1 reference

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Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

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Alzheimer's disease: a re-examination of the amyloid hypothesis

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(D-Ala2)-Met-enkephalinamide: a potent, long-lasting synthetic pentapeptide analgesic

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Glutamate release in severe brain ischaemia is mainly by reversed uptake.

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Biochemistry of altered brain proteins in Alzheimer's disease

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Neuritic plaque amyloid in Alzheimer's disease is highly racemized

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Aging is associated with a decrease in synaptosomal glutamate uptake and an increase in the susceptibility of synaptosomal vitamin E to oxidative stress.

1 reference

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Occurrence of neuropathological changes and dementia of Alzheimer's disease in Down's syndrome.

1 reference

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Identifiers

10.1016/S0306-4522(01)00155-5

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 November 2017

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