(Q43764329)

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Inhibition of calpain-mediated apoptosis by E-64 d-reduced immediate early gene (IEG) expression and reactive astrogliosis in the lesion and penumbra following spinal cord injury in rats

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Europe PubMed Central

PubMed publication ID

26 November 2017

Inhibition of calpain-mediated apoptosis by E-64 d-reduced immediate early gene (IEG) expression and reactive astrogliosis in the lesion and penumbra following spinal cord injury in rats (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

immediate early gene

1 reference

based on heuristic

inferred from title

author name string

Ray SK

1

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Matzelle DD

2

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Wilford GG

3

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Hogan EL

4

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Banik NL

5

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

language of work or name

0 references

publication date

1 October 2001

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

916

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

1-2

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

115-126

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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Review of current evidence for apoptosis after spinal cord injury.

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Apoptosis: overview and signal transduction pathways

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Calpain: a protease in search of a function?

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Calcium-activated neutral proteinase in rat brain myelin and subcellular fractions

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Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia.

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Calcium-activated neutral protease (calpain) system: structure, function, and regulation

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Immediate-early gene protein expression in neurons undergoing delayed death, but not necrosis, following hypoxic-ischaemic injury to the young rat brain

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Induction of c-fos mRNA and protein in neurons and glia after traumatic brain injury: pharmacological characterization.

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Calcium influx and activation of calpain I mediate acute reactive gliosis in injured spinal cord

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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The glial scar and central nervous system repair

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Isolation of cDNA clones encoding rat glial fibrillary acidic protein: expression in astrocytes and in Schwann cells

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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Ca2+-accumulation in experimental spinal cord trauma

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N-methyl-D-aspartate receptors mediate activation of the c-fos proto-oncogene in a model of brain injury.

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Constitutive apoptosis in human neutrophils requires synergy between calpains and the proteasome downstream of caspases

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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Apoptosis of oligodendrocytes occurs for long distances away from the primary injury after compression trauma to rat spinal cord

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Neuronal and glial apoptosis after traumatic spinal cord injury.

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Analysis of a segment of the human glial fibrillary acidic protein gene that directs astrocyte-specific transcription

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Inhibition of calpain in intact platelets by the thiol protease inhibitor E-64d

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Reduction of neurite outgrowth in a model of glial scarring following CNS injury is correlated with the expression of inhibitory molecules on reactive astrocytes

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7 January 2021

Validation of the weight-drop contusion model in rats: a comparative study of human spinal cord injury

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Motoneurons deprived of trophic support in vitro require new gene expression to undergo programmed cell death.

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Stimulus-transcription coupling in the nervous system: involvement of the inducible proto-oncogenes fos and jun.

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Release of mitochondrial cytochrome c and DNA fragmentation after cold injury-induced brain trauma in mice: possible role in neuronal apoptosis.

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Sensitivity limitations encountered in enhanced horseradish peroxidase catalysed chemiluminescence

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Calpain-calpastatin interactions in epidermoid carcinoma KB cells

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Fragmentation of an endogenous inhibitor upon complex formation with high- and low-Ca2+-requiring forms of calcium-activated neutral proteases

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Non-erythroid alpha-spectrin breakdown by calpain and interleukin 1 beta-converting-enzyme-like protease(s) in apoptotic cells: contributory roles of both protease families in neuronal apoptosis

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Differential expression of the immediate early genes c-fos, c-jun, junB, and NGFI-B in the rat brain following transient forebrain ischemia.

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Regional calpain and caspase-3 proteolysis of alpha-spectrin after traumatic brain injury

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Emerging roles of caspase-3 in apoptosis

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Oxidative stress and Ca2+ influx upregulate calpain and induce apoptosis in PC12 cells

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E-64-d prevents both calpain upregulation and apoptosis in the lesion and penumbra following spinal cord injury in rats

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Pretreatment with Calpain Inhibitor CEP-4143 Inhibits Calpain I Activation and Cytoskeletal Degradation, Improves Neurological Function, and Enhances Axonal Survival After Traumatic Spinal Cord Injury

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Continuous c-fos expression precedes programmed cell death in vivo

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Calpain activation in apoptosis

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Distinct caspase cascades are initiated in receptor-mediated and chemical-induced apoptosis.

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Signal transduction in neuronal death.

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Caspase-3 is activated following axotomy of neonatal facial motoneurons and caspase-3 gene deletion delays axotomy-induced cell death in rodents

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Caspase-mediated fragmentation of calpain inhibitor protein calpastatin during apoptosis

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Calpain activation is upstream of caspases in radiation-induced apoptosis.

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Chromatin cleavage in apoptosis: association with condensed chromatin morphology and dependence on macromolecular synthesis.

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Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury.

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Apoptosis in cellular compartments of rat spinal cord after severe contusion injury

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Involvement of cellular proteolytic machinery in apoptosis

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Identifiers

10.1016/S0006-8993(01)02874-8

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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