(Q43786456)

English

Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence

scientific article published in November 2001

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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author name string

Shen KF

1

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Crain SM

2

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Europe PubMed Central

PubMed publication ID

26 November 2017

language of work or name

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publication date

1 November 2001

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Europe PubMed Central

PubMed publication ID

26 November 2017

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Europe PubMed Central

PubMed publication ID

26 November 2017

919

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

20-30

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

Selective modulation of 3H-spiperone labeled 5-HT receptors by subchronic treatment with the ganglioside GM1 in the rat

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Kappa opioid receptors participate in nerve growth factor-induced hyperalgesia

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Inhibition of the antianalgesic action of dynorphin A in mice by cholera toxin

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Possible role of gangliosides in regulating an adenylate cyclase-linked 5-hydroxytryptamine (5-HT1) receptor.

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Mu-opioid receptor desensitization by beta-arrestin-2 determines morphine tolerance but not dependence

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Mechanism of adenylate cyclase activation by cholera toxin: Inhibition of GTP hydrolysis at the regulatory site

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Maturation of opioid sensitivity of fetal mouse dorsal root ganglion neuron perikarya in organotypic cultures: regulation by spinal cord

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Inhibitor of cyclic AMP-dependent protein kinase blocks opioid-induced prolongation of the action potential of mouse sensory ganglion neurons in dissociated cell cultures

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Opioids can evoke direct receptor-mediated excitatory effects on sensory neurons

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After chronic opioid exposure sensory neurons become supersensitive to the excitatory effects of opioid agonists and antagonists as occurs after acute elevation of GM1 ganglioside

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Ultra-low concentrations of naloxone selectively antagonize excitatory effects of morphine on sensory neurons, thereby increasing its antinociceptive potency and attenuating tolerance/dependence during chronic cotreatment

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7 January 2021

GM1 ganglioside-induced modulation of opioid receptor-mediated functions

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7 January 2021

Modulation of opioid analgesia, tolerance and dependence by Gs-coupled, GM1 ganglioside-regulated opioid receptor functions

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Enhanced analgesic potency and reduced tolerance of morphine in 129/SvEv mice: evidence for a deficiency in GM1 ganglioside-regulated excitatory opioid receptor functions

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Antagonists of excitatory opioid receptor functions enhance morphine's analgesic potency and attenuate opioid tolerance/dependence liability

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Acute thermal hyperalgesia elicited by low-dose morphine in normal mice is blocked by ultra-low-dose naltrexone, unmasking potent opioid analgesia

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Pertussis toxin blocks depressant effects of opioid, monoaminergic and muscarinic agonists on dorsal-horn network responses in spinal cord-ganglion cultures

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Direct coupling of opioid receptors to both stimulatory and inhibitory guanine nucleotide-binding proteins in F-11 neuroblastoma-sensory neuron hybrid cells

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Endogenous GM1 ganglioside of the plasma membrane promotes neuritogenesis by two mechanisms

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Promotion of neuronal survival by GM1 ganglioside. Phenomenology and mechanism of action.

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Role of membrane gangliosides in the binding and action of bacterial toxins.

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Recent advances in identifying the functions of gangliosides

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ADP-ribosylation of membrane proteins catalyzed by cholera toxin: basis of the activation of adenylate cyclase

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7 January 2021

G proteins and dual control of adenylate cyclase

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7 January 2021

Activators of adenylate cyclase and cyclic AMP prolong calcium-dependent action potentials of mouse sensory neurons in culture by reducing a voltage-dependent potassium conductance

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7 January 2021

Coordinated assembly of multisubunit proteins: oligomerization of bacterial enterotoxins in vivo and in vitro

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7 January 2021

Opioid delta-receptor involvement in supraspinal and spinal antinociception in mice

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Cholera toxin and cholera B subunit as oral-mucosal adjuvant and antigen vector systems

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Spinal amino acid release and precipitated withdrawal in rats chronically infused with spinal morphine

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Morphine analgesia and acute physical dependence: rapid onset of two opposing, dose-related processes

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The ionic channels formed by cholera toxin in planar bilayer lipid membranes are entirely attributable to its B-subunit

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TOLERANCE TO AND PHYSICAL DEPENDENCE ON MORPHINE IN RATS

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Cholera toxin genes: nucleotide sequence, deletion analysis and vaccine development

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Mechanism of action of choleragen andE. coli heat-labile enterotoxin: activation of adenylate cyclase by ADP-ribosylation

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Multiple Mechanisms of withdrawal from Opioid Drugs

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Three-dimensional structure of cholera toxin penetrating a lipid membrane

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Recombinant system for overexpression of cholera toxin B subunit in Vibrio cholerae as a basis for vaccine development

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Cholera toxin and pertussis toxin on opioid- and α2-mediated supraspinal analgesia in mice

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Intracerebroventricular injection of antibodies directed against Gs alpha enhances the supraspinal antinociception induced by morphine, beta-endorphin and clonidine in mice

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Exogenous gangliosides. How do they cross the blood-brain barrier and how do they inhibit cell proliferation.

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Dual opioid modulation of the action potential duration of mouse dorsal root ganglion neurons in culture

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Cholera toxin-A subunit blocks opioid excitatory effects on sensory neuron action potentials indicating mediation by Gs-linked opioid receptors

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Cholera toxin-B subunit blocks excitatory effects of opioids on sensory neuron action potentials indicating that GM1 ganglioside may regulate Gs-linked opioid receptor functions

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Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid 'dependence' without tolerance

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Ultra-low doses of naltrexone or etorphine increase morphine's antinociceptive potency and attenuate tolerance/dependence in mice.

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Brief treatment of sensory ganglion neurons with GM1 ganglioside enhances the efficacy of opioid excitatory effects on the action potential

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Hyperalgesia during withdrawal as a means of measuring the degree of dependence in morphine dependent rats

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Inhibition of a cAMP-dependent Ca-activated K conductance by forskolin prolongs Ca action potential duration in lamprey sensory neurons

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Interaction of the delta-opioid receptor with GM1 ganglioside: conversion from inhibitory to excitatory mode

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The role of GM1 ganglioside in regulating excitatory opioid effects

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Reduction in oral immunogenicity of cholera toxin B subunit by N-terminal peptide addition.

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based on heuristic

inferred from DOI database lookup

Differential kinetics and distribution of antibodies in serum and nasal and vaginal secretions after nasal and oral vaccination of humans

1 reference

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based on heuristic

inferred from DOI database lookup

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10.1016/S0006-8993(01)02990-0

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Europe PubMed Central

PubMed publication ID

26 November 2017

release_hie6ehw3dbdmfllcvzp3w5lm6y

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https://api.fatcat.wiki/v0/release/hie6ehw3dbdmfllcvzp3w5lm6y

24 November 2022

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 November 2017

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