(Q43796816)

26 November 2017

GABA(B) receptor activation enhances mGluR-mediated responses at cerebellar excitatory synapses (English)

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26 November 2017

1

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26 November 2017

Yoshioka T

2

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26 November 2017

Konishi S

series ordinal

3

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26 November 2017

language of work or name

English

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publication date

1 December 2001

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26 November 2017

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26 November 2017

4

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26 November 2017

1207-1216

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26 November 2017

12

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https://scigraph.springernature.com/pub.10.1038/nn764

26 November 2017

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cites work

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Immunolocalization of the mGluR1b splice variant of the metabotropic glutamate receptor 1 at parallel fiber-Purkinje cell synapses in the rat cerebellar cortex

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Responses to Metabotropic Glutamate Receptor Activation in Cerebellar Purkinje Cells: Induction of an Inward Current.

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Trans-ACPD, a metabotropic receptor agonist, produces calcium mobilization and an inward current in cultured cerebellar Purkinje neurons

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Adenosine Receptors: Autoradiographic Evidence for Their Location on Axon Terminals of Excitatory Neurons

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Light-microscopic distribution and parasagittal organisation of muscarinic receptors in rabbit cerebellar cortex

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Monoaminergic long-term facilitation of GABA-mediated inhibitory transmission at cerebellar synapses.

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GABA(B) receptor-mediated presynaptic inhibition of glutamatergic and GABAergic transmission in the basolateral amygdala

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Pre- and postsynaptic determinants of EPSC waveform at cerebellar climbing fiber and parallel fiber to Purkinje cell synapses.

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Contributions of calcium-dependent and calcium-independent mechanisms to presynaptic inhibition at a cerebellar synapse.

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Opioids mobilize calcium from inositol 1,4,5-trisphosphate-sensitive stores in NG108-15 cells

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Cerebellar GABA B receptors modulate function of GABA A receptors

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Postsynaptic current mediated by metabotropic glutamate receptors in cerebellar Purkinje cells.

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Molecular machines integrate coincident synaptic signals

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Local calcium release in dendritic spines required for long-term synaptic depression

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GABAB autoreceptors regulate the induction of LTP

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Facilitation of the induction of long-term potentiation by GABAB receptors.

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GABAB receptors, monoamine receptors, and postsynaptic inositol trisphosphate-induced Ca2+ release are involved in the induction of long-term potentiation at visual cortical inhibitory synapses.

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Receptors for dopamine and somatostatin: formation of hetero-oligomers with enhanced functional activity

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Direct protein-protein coupling enables cross-talk between dopamine D5 and gamma-aminobutyric acid A receptors

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State-dependent cross-inhibition between transmitter-gated cation channels.

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7 January 2021

Identifiers

DOI

10.1038/NN764

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Dimensions Publication ID

26 November 2017

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