(Q43806443)

27 November 2017

title

Phenotypic effects of expanded ataxin-1 polyglutamines with interruptions in vitro (English)

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27 November 2017

V Calabresi

1

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27 November 2017

S Guida

series ordinal

2

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27 November 2017

A Servadio

series ordinal

3

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27 November 2017

C Jodice

series ordinal

4

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27 November 2017

publication date

1 October 2001

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27 November 2017

published in

Brain Research Bulletin

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27 November 2017

volume

56

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27 November 2017

issue

3-4

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27 November 2017

page(s)

337-342

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Transgenic models of Huntington's disease

27 November 2017

cites work

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7 January 2021

Gametic and somatic tissue-specific heterogeneity of the expanded SCA1 CAG repeat in spinocerebellar ataxia type 1.

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7 January 2021

Evidence for a mechanism predisposing to intergenerational CAG repeat instability in spinocerebellar ataxia type I.

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7 January 2021

Identification of genes that modify ataxin-1-induced neurodegeneration.

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7 January 2021

CAG repeat instability, cryptic sequence variation and pathogeneticity: evidence from different loci

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7 January 2021

Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice

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7 January 2021

Expansion of an unstable trinucleotide CAG repeat in spinocerebellar ataxia type 1.

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7 January 2021

Interruptions in the Triplet Repeats of SCA1 and FRAXA Reduce the Propensity and Complexity of Slipped Strand DNA (S-DNA) Formation†

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https://api.crossref.org/works/10.1016%2FS0361-9230%2801%2900600-1

7 January 2021

Glutamine repeats as polar zippers: their possible role in inherited neurodegenerative diseases

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7 January 2021

Glutamine repeats and inherited neurodegenerative diseases: molecular aspects

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7 January 2021

A novel CAG repeat configuration in the SCA1 gene: implications for the molecular diagnostics of spinocerebellar ataxia type 1

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7 January 2021

Intranuclear neuronal inclusions: a common pathogenic mechanism for glutamine-repeat neurodegenerative diseases?

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7 January 2021

Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

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7 January 2021

Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo

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7 January 2021

Self-assembly of polyglutamine-containing huntingtin fragments into amyloid-like fibrils: implications for Huntington's disease pathology

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7 January 2021

Expression analysis of the ataxin-1 protein in tissues from normal and spinocerebellar ataxia type 1 individuals

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7 January 2021

Ataxin-1 with an expanded glutamine tract alters nuclear matrix-associated structures

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7 January 2021

Single-step purification of polypeptides expressed in Escherichia coli as fusions with glutathione S-transferase

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7 January 2021

Polyglutamine expansion as a pathological epitope in Huntington's disease and four dominant cerebellar ataxias

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7 January 2021

Huntingtin is required for neurogenesis and is not impaired by the Huntington's disease CAG expansion

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7 January 2021

Dynamic mutations hit double figures

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7 January 2021

Spinocerebellar ataxia: variable age of onset and linkage to human leukocyte antigen in a large kindred.

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7 January 2021

Glutamine repeats and neurodegeneration

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https://api.crossref.org/works/10.1016%2FS0361-9230%2801%2900600-1

7 January 2021

10.1016/S0361-9230(01)00600-1

Identifiers

DOI

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27 November 2017

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