(Q43822967)

English

Intracellular acidification by inhibition of the Na+/H+-exchanger leads to caspase-independent death of cerebellar granule neurons resembling paraptosis.

scientific article published in July 2004

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scholarly article

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25 December 2019

Intracellular acidification by inhibition of the Na+/H+-exchanger leads to caspase-independent death of cerebellar granule neurons resembling paraptosis (English)

1 reference

Europe PubMed Central

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25 December 2019

Jörg B. Schulz

7

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25 December 2019

author name string

D Schneider

1

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25 December 2019

E Gerhardt

2

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25 December 2019

J Bock

3

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25 December 2019

M M Müller

4

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25 December 2019

H Wolburg

5

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25 December 2019

F Lang

6

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25 December 2019

publication date

1 July 2004

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25 December 2019

Cell Death & Differentiation

1 reference

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25 December 2019

11

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25 December 2019

7

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25 December 2019

760-770

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25 December 2019

https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401377

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7 January 2021

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Phosphorus-31 magnetic resonance spectra reveal prolonged intracellular acidosis in the brain following subarachnoid hemorrhage

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Maintenance of caspase-3 proenzyme dormancy by an intrinsic "safety catch" regulatory tripeptide

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Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

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The expanding family of eucaryotic Na(+)/H(+) exchangers

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SM-20220, a Na(+)/H(+) exchanger inhibitor: effects on ischemic brain damage through edema and neutrophil accumulation in a rat middle cerebral artery occlusion model.

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The Na(+)/H(+) exchanger SM-20220 attenuates ischemic injury in in vitro and in vivo models

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7 January 2021

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Inhibition of Na(+)/H(+) exchange by SM-20220 attenuates free fatty acid efflux in rat cerebral cortex during ischemia-reperfusion injury

1 reference

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7 January 2021

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Depolarization or glutamate receptor activation blocks apoptotic cell death of cultured cerebellar granule neurons

1 reference

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7 January 2021

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Potassium deprivation-induced apoptosis of cerebellar granule neurons: a sequential requirement for new mRNA and protein synthesis, ICE-like protease activity, and reactive oxygen species.

1 reference

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7 January 2021

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Apoptosis in cerebellar granule cells is blocked by high KCl, forskolin, and IGF-1 through distinct mechanisms of action: the involvement of intracellular calcium and RNA synthesis

1 reference

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7 January 2021

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Induction of apoptosis in cerebellar granule neurons by low potassium: inhibition of death by insulin-like growth factor I and cAMP.

1 reference

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7 January 2021

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Mediation of neuronal apoptosis by enhancement of outward potassium current.

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7 January 2021

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Role of the outward delayed rectifier K+ current in ceramide-induced caspase activation and apoptosis in cultured cortical neurons.

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NMDA receptor-mediated K+ efflux and neuronal apoptosis.

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Enhancement of outward potassium current may participate in beta-amyloid peptide-induced cortical neuronal death.

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Functional significance of cell volume regulatory mechanisms.

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Hydrogen Ion Regulation in Rat Cerebellar Granule Cells Studied by Single-Cell Fluorescence Microscopy

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7 January 2021

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Intracellular pH regulation in cultured mouse oligodendrocytes

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7 January 2021

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Mechanisms of pHi regulation studied in individual neurons cultured from mouse cerebral cortex

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7 January 2021

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Activation of the CED3/ICE-related protease CPP32 in cerebellar granule neurons undergoing apoptosis but not necrosis

1 reference

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7 January 2021

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Cascade of caspase activation in potassium-deprived cerebellar granule neurons: targets for treatment with peptide and protein inhibitors of apoptosis

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7 January 2021

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Insulin-like growth factor-1-mediated protection from neuronal apoptosis is linked to phosphorylation of the pro-apoptotic protein BAD but not to inhibition of cytochrome c translocation in rat cerebellar neurons

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Caspase-3 expression by cerebellar granule neurons is regulated by calcium and cyclic AMP.

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7 January 2021

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Apoptosis-inducing factor is involved in the regulation of caspase-independent neuronal cell death

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Four deaths and a funeral: from caspases to alternative mechanisms.

1 reference

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7 January 2021

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Apoptosis in caspase-inhibited neurons

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Living T9 glioma cells expressing membrane macrophage colony-stimulating factor produce immediate tumor destruction by polymorphonuclear leukocytes and macrophages via a "paraptosis"-induced pathway that promotes systemic immunity against intracrani

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Identifiers

10.1038/SJ.CDD.4401377

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15017383%20AND%20SRC:MED&resulttype=core&format=json

25 December 2019

Dimensions Publication ID

0 references

1 reference

Europe PubMed Central

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25 December 2019

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