(Q43960640)

English

Impairing the amphetamine conditioning in rats through the inhibition of hippocampal calcium/calmodulin-dependent protein kinase II activity

scientific article published in March 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Impairing the amphetamine conditioning in rats through the inhibition of hippocampal calcium/calmodulin-dependent protein kinase II activity (English)

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

author name string

Tan SE

1

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

publication date

1 March 2002

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Neuropharmacology

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

42

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

540-547

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Amphetamine-induced behavior, dopamine release, and c-fos mRNA expression: modulation by environmental novelty.

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Independence of amphetamine reward from locomotor stimulation demonstrated by conditioned place preference.

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Protein kinases A and C are involved in the mechanisms underlying consolidation of cocaine place conditioning.

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Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats

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Amphetamine increases the phosphorylation of neuromodulin and synapsin I in rat striatal synaptosomes.

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Modality-specific retrograde amnesia of fear

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Cellular and molecular mechanisms of drug dependence

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Effects of the AMPA/kainate receptor antagonist DNQX in the nucleus accumbens on drug-induced conditioned place preference

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Amphetamine enhances memory retention and facilitates norepinephrine release from the hippocampus in rats

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Chemical stimulation of the ventral hippocampus elevates nucleus accumbens dopamine by activating dopaminergic neurons of the ventral tegmental area.

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Learning mechanisms: the case for CaM-KII.

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Animal models of drug craving

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Control of memory formation through regulated expression of a CaMKII transgene

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The nucleus accumbens as a complex of functionally distinct neuronal ensembles: an integration of behavioural, electrophysiological and anatomical data.

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Repeated cocaine modifies the mechanism by which amphetamine releases dopamine.

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Ibotenic acid lesions of the dorsal prefrontal cortex disrupt the expression of behavioral sensitization to cocaine.

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Biochemical adaptations in the mesolimbic dopamine system in response to heroin self-administration.

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based on heuristic

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based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/S0028-3908(02)00004-7

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

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