(Q43991985)

28 November 2017

title

Mitochondrial permeability transition and cytochrome c release induced by selenite (English)

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28 November 2017

Yushan Zhu

1

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28 November 2017

Huibi Xu

series ordinal

2

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28 November 2017

Kaixun Huang

series ordinal

3

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28 November 2017

publication date

1 May 2002

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Journal of Inorganic Biochemistry

28 November 2017

published in

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28 November 2017

volume

90

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28 November 2017

issue

1-2

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28 November 2017

page(s)

43-50

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From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state

28 November 2017

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Mitochondrial calcium uptake from physiological-type pulses of calcium. A description of the rapid uptake mode

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Prooxidants open both the mitochondrial permeability transition pore and a low-conductance channel in the inner mitochondrial membrane

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux

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7 January 2021

A simplification of the protein assay method of Lowry et al. which is more generally applicable

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7 January 2021

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

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7 January 2021

Release of cytochrome c from heart mitochondria is induced by high Ca2+ and peroxynitrite and is responsible for Ca(2+)-induced inhibition of substrate oxidation.

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Circulating soluble vascular adhesion protein 1 accounts for the increased serum monoamine oxidase activity in chronic liver disease

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7 January 2021

Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

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7 January 2021

Regulation of the permeability transition pore in skeletal muscle mitochondria. Modulation By electron flow through the respiratory chain complex i.

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The Mitochondrial F0F1-ATPase proton pump is required for function of the proapoptotic protein Bax in yeast and mammalian cells.

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Ceramide Induces CytochromecRelease from Isolated Mitochondria

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7 January 2021

Selenium: potent stimulator of tyrosyl phosphorylation and activator of MAP kinase

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7 January 2021

Selenite negatively regulates caspase-3 through a redox mechanism

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7 January 2021

Effect of selenium compounds and thiols on human mammary tumor cells

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7 January 2021

Comparison of the effects of an organic and an inorganic form of selenium on a mammary carcinoma cell line

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7 January 2021

Inhibition of selenite-catalyzed superoxide generation and formation of elemental selenium (Se(o)) by copper, zinc, and aurintricarboxylic acid (ATA)

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7 January 2021

Induction of differentiation and apoptosis by sodium selenite in human colonic carcinoma cells (HT29)

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7 January 2021

Induction of the mitochondrial permeability transition causes release of the apoptogenic factor cytochrome c

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7 January 2021

Mitochondria and apoptosis

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7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

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7 January 2021

The importance of selenium to human health

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7 January 2021

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Identifiers

DOI

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28 November 2017

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