(Q44011679)

English

Increased transforming growth factor-beta(1) circulating levels and production in human monocytes after 3-hydroxy-3-methyl-glutaryl-coenzyme a reductase inhibition with pravastatin

scientific article published in June 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Increased transforming growth factor-beta(1) circulating levels and production in human monocytes after 3-hydroxy-3-methyl-glutaryl-coenzyme a reductase inhibition with pravastatin (English)

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

author name string

Ettore Porreca

1

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Concetta Di Febbo

2

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Giovanna Baccante

3

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Marcello Di Nisio

4

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Franco Cuccurullo

5

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

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publication date

1 June 2002

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Europe PubMed Central

PubMed publication ID

28 November 2017

Journal of the American College of Cardiology

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Europe PubMed Central

PubMed publication ID

28 November 2017

39

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Europe PubMed Central

PubMed publication ID

28 November 2017

11

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

1752-1757

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

Transforming growth factor-beta: recent progress and new challenges

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The pathogenesis of atherosclerosis: a perspective for the 1990s

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Decreased type II/type I TGF-beta receptor ratio in cells derived from human atherosclerotic lesions. Conversion from an antiproliferative to profibrotic response to TGF-beta1.

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Beta ig-h3, a transforming growth factor-beta-inducible gene, is overexpressed in atherosclerotic and restenotic human vascular lesions

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Expression of transforming growth factor-beta 1 is increased in human vascular restenosis lesions

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Atherosclerosis: coagulation and fibrinolysis

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Active and acid-activatable TGF-beta in human sera, platelets and plasma

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Upregulation of endothelial nitric oxide synthase by HMG CoA reductase inhibitors

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HMG-CoA Reductase Inhibitors Decrease CD11b Expression and CD11b-Dependent Adhesion of Monocytes to Endothelium and Reduce Increased Adhesiveness of Monocytes Isolated From Patients With Hypercholesterolemia

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Inhibition of proinflammatory cytokine production by pravastatin

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Simvastatin inhibits the monocyte expression of proinflammatory cytokines in patients with hypercholesterolemia

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Release and activation of platelet latent TGF–β in blood clots during dissolution with plasmin

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Increased transforming growth factor-beta production and gene expression by peripheral blood monocytes of hypertensive patients

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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NK-104, a newly developed HMG-CoA reductase inhibitor, suppresses neointimal thickening by inhibiting smooth muscle cell growth and fibronectin production in balloon-injured rabbit carotid artery

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3-Hydroxy-3-methylglutaryl CoA reductase inhibitors up-regulate transforming growth factor-beta signaling in cultured heart cells via inhibition of geranylgeranylation of RhoA GTPase

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Regulation of the mevalonate pathway

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Beta-type transforming growth factor specifies organizational behavior in vascular smooth muscle cell cultures

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Direct transfer of transforming growth factor beta 1 gene into arteries stimulates fibrocellular hyperplasia

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Identifiers

10.1016/S0735-1097(02)01857-0

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

28 November 2017

ResearchGate publication ID

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