(Q44041831)

29 November 2017

title

Acrolein causes transcriptional induction of phase II genes by activation of Nrf2 in human lung type II epithelial (A549) cells (English)

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29 November 2017

R Tirumalai

1

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29 November 2017

T Rajesh Kumar

series ordinal

2

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29 November 2017

Kim Hue Mai

series ordinal

3

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29 November 2017

Shyam Biswal

series ordinal

4

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29 November 2017

publication date

1 June 2002

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29 November 2017

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29 November 2017

volume

132

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29 November 2017

issue

1

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29 November 2017

page(s)

27-36

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Nrf2, a Cap'n'Collar transcription factor, regulates induction of the heme oxygenase-1 gene

29 November 2017

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Inhibition of cell proliferation and AP-1 activity by acrolein in human A549 lung adenocarcinoma cells due to thiol imbalance and covalent modifications

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Acrolein causes inhibitor kappaB-independent decreases in nuclear factor kappaB activation in human lung adenocarcinoma (A549) cells

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Phosphatidylinositol 3-kinase, not extracellular signal-regulated kinase, regulates activation of the antioxidant-responsive element in IMR-32 human neuroblastoma cells

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Nrf2-dependent activation of the antioxidant responsive element by tert-butylhydroquinone is independent of oxidative stress in IMR-32 human neuroblastoma cells

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Lipid peroxidation end products-responded induction of a preneoplastic marker enzyme glutathione S-transferase P-form (GST-P) in rat liver on admistration via the portal vein

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Evidence of a defective thiol status of alveolar macrophages from COPD patients and smokers. Chronic obstructive pulmonary disease

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7 January 2021

Oxidative stress promotes the development of transformation: involvement of a potent mutagenic lipid peroxidation product, acrolein

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Acrolein is a product of lipid peroxidation reaction. Formation of free acrolein and its conjugate with lysine residues in oxidized low density lipoproteins

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Identifiers

DOI

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29 November 2017

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