(Q44057388)

29 November 2017

title

Mitochondrial permeability transition can be directly monitored in living neurons (English)

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29 November 2017

Gillessen T

1

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29 November 2017

Grasshoff C

series ordinal

2

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29 November 2017

Szinicz L

series ordinal

3

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29 November 2017

publication date

1 June 2002

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Biomedicine and Pharmacotherapy

29 November 2017

published in

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29 November 2017

volume

56

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29 November 2017

issue

4

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29 November 2017

page(s)

186-193

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Mitochondrial and extramitochondrial apoptotic signaling pathways in cerebrocortical neurons

29 November 2017

cites work

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Chaotropic agents and increased matrix volume enhance binding of mitochondrial cyclophilin to the inner mitochondrial membrane and sensitize the mitochondrial permeability transition to [Ca2+]

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7 January 2021

The cytoskeleton of digitonin-treated rat hepatocytes

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7 January 2021

Imaging the permeability pore transition in single mitochondria

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https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Ca(2+)-induced mitochondrial membrane permeabilization: role of coenzyme Q redox state

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https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Ca(2+)-permeable AMPA/kainate and NMDA channels: high rate of Ca2+ influx underlies potent induction of injury.

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7 January 2021

Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

1 reference

https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

1 reference

https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Imaging the mitochondrial permeability transition pore in intact cells.

1 reference

https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

The quantitative retention of cholesterol in mouse liver prepared for electron microscopy by fixation in a digitonin-containing aldehyde solution

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7 January 2021

Mitochondrial dysfunction is a primary event in glutamate neurotoxicity

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https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Cortical neurones exhibiting kainate-activated Co2+ uptake are selectively vulnerable to AMPA/kainate receptor-mediated toxicity

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https://api.crossref.org/works/10.1016%2FS0753-3322%2802%2900184-1

7 January 2021

Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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7 January 2021

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Identifiers

DOI

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29 November 2017

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