(Q44070700)

29 November 2017

title

Modulation of gene expression in transgenic mouse hearts overexpressing calsequestrin (English)

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29 November 2017

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Ihara Y

1

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29 November 2017

Suzuki YJ

2

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29 November 2017

Kitta K

3

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29 November 2017

Jones LR

4

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29 November 2017

Ikeda T

5

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29 November 2017

publication date

1 July 2002

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29 November 2017

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29 November 2017

volume

32

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29 November 2017

issue

1

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29 November 2017

page(s)

21-29

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Left ventricular hypertrophy and mortality--results from the Framingham Study

29 November 2017

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Complex formation between junctin, triadin, calsequestrin, and the ryanodine receptor. Proteins of the cardiac junctional sarcoplasmic reticulum membrane

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Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin

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Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic mice

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Modulation of focal and global Ca2+ release in calsequestrin-overexpressing mouse cardiomyocytes

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Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression

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Remodelling of ionic currents in hypertrophied and failing hearts of transgenic mice overexpressing calsequestrin

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Cardiac beta ARK1 inhibition prolongs survival and augments beta blocker therapy in a mouse model of severe heart failure

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Regulation of GATA-4 and AP-1 in transgenic mice overexpressing cardiac calsequestrin

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Remodelling of Cardiac Extracellular Matrix duringβ-adrenergic Stimulation: Upregulation of SPARC in the Myocardium of Adult Rats,

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Unchanged Protein Levels of SERCA II and Phospholamban but Reduced Ca 2+ Uptake and Ca 2+ -ATPase Activity of Cardiac Sarcoplasmic Reticulum From Dilated Cardiomyopathy Patients Compared With Patients With Nonfailing Hearts

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7 January 2021

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7 January 2021

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7 January 2021

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Molecular mechanisms of doxorubicin-induced cardiomyopathy. Selective suppression of Reiske iron-sulfur protein, ADP/ATP translocase, and phosphofructokinase genes is associated with ATP depletion in rat cardiomyocytes

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A mouse model for mitochondrial myopathy and cardiomyopathy resulting from a deficiency in the heart/muscle isoform of the adenine nucleotide translocator

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7 January 2021

10.1016/S0143-4160(02)00096-9

Identifiers

DOI

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29 November 2017

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