(Q44095011)

English

Lead alters parathyroid hormone-related peptide and transforming growth factor-beta1 effects and AP-1 and NF-kappaB signaling in chondrocytes

scientific article published in July 2002

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Lead alters parathyroid hormone-related peptide and transforming growth factor-beta1 effects and AP-1 and NF-kappaB signaling in chondrocytes (English)

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

1 reference

based on heuristic

inferred from title

author name string

Michael J Zuscik

1

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Dhruv B Pateder

2

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

J Edward Puzas

3

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Edward M Schwarz

4

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Randy N Rosier

5

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Regis J O'Keefe

6

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

publication date

1 July 2002

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Journal of Orthopaedic Research

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

20

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

811-818

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

Selective vulnerability of the developing brain to lead

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Current issues in the epidemiology and toxicology of occupational exposure to lead

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Parathyroid hormone causes translocation of protein kinase-C from cytosol to membranes in rat osteosarcoma cells

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TGF-beta 1 prevents hypertrophy of epiphyseal chondrocytes: regulation of gene expression for cartilage matrix proteins and metalloproteases

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Terminal differentiation of chondrocytes in culture is a spontaneous process and is arrested by transforming growth factor-beta 2 and basic fibroblast growth factor in synergy

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Differential effects of parathyroid hormone on chick growth plate and articular chondrocytes

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Bone morphogenetic protein signaling is required for maintenance of differentiated phenotype, control of proliferation, and hypertrophy in chondrocytes

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The production of transforming growth factor-beta by chick growth plate chondrocytes in short term monolayer culture

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Effect of lead on bone and cartilage in sexually mature rats: a morphometric and histomorphometry study

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BMP-6 is an autocrine stimulator of chondrocyte differentiation

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Bone morphogenetic protein-7 in growth-plate chondrocytes: regulation by retinoic acid is dependent on the stage of chondrocyte maturation

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Effects of lead exposure on skeletal development in rats

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End labeling studies of fragmented DNA in the avian growth plate: evidence of apoptosis in terminally differentiated chondrocytes

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Effects of lead on growth plate chondrocyte phenotype

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Responsiveness to retinoic acid changes during chondrocyte maturation.

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Lethal skeletal dysplasia from targeted disruption of the parathyroid hormone-related peptide gene

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Trace elements in regulation of NF-kappaB activity

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Gene expression of neurotrophins and their receptors in lead nitrate-induced rat liver hyperplasia

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Effects of transforming growth factor-beta 1 and fibroblast growth factor on DNA synthesis in growth plate chondrocytes are enhanced by insulin-like growth factor-I

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Effects of transforming growth factor-beta on matrix synthesis by chick growth plate chondrocytes

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Metal ion selectivity for formation of the calmodulin-metal-target peptide ternary complex studied by surface plasmon resonance spectroscopy.

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PTHrP expression in chondrocytes, regulation by TGF-beta, and interactions between epiphyseal and growth plate chondrocytes

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Radiographic findings in congenital lead poisoning

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Lead inhibits meso-2,3-dimercaptosuccinic acid induced calcium transients in cultured rhesus monkey kidney cells.

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Lead activates nuclear transcription factor-kappaB, activator protein-1, and amino-terminal c-Jun kinase in pheochromocytoma cells

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Expression of a truncated, kinase-defective TGF-beta type II receptor in mouse skeletal tissue promotes terminal chondrocyte differentiation and osteoarthritis

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Parathyroid hormone-related peptide (PTHrP)-dependent and -independent effects of transforming growth factor beta (TGF-beta) on endochondral bone formation

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Target Organs The Blood*

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Identifiers

10.1016/S0736-0266(02)00007-4

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 November 2017

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