(Q44126738)

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Activation of mitogen-activated protein kinases (MAPKs) by aromatic hydrocarbons: role in the regulation of aryl hydrocarbon receptor (AHR) function

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Europe PubMed Central

PubMed publication ID

30 November 2017

Activation of mitogen-activated protein kinases (MAPKs) by aromatic hydrocarbons: role in the regulation of aryl hydrocarbon receptor (AHR) function (English)

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Europe PubMed Central

PubMed publication ID

30 November 2017

author name string

Zongqing Tan

1

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Europe PubMed Central

PubMed publication ID

30 November 2017

Xiaoqing Chang

2

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Europe PubMed Central

PubMed publication ID

30 November 2017

Alvaro Puga

3

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Europe PubMed Central

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30 November 2017

Ying Xia

4

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30 November 2017

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publication date

1 September 2002

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30 November 2017

Biochemical Pharmacology

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30 November 2017

64

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Europe PubMed Central

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30 November 2017

5-6

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Europe PubMed Central

PubMed publication ID

30 November 2017

771-780

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Europe PubMed Central

PubMed publication ID

30 November 2017

The aryl hydrocarbon receptor: A comparative perspective

1 reference

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Induction of cytochrome P4501A1.

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Aryl-hydrocarbon receptor-deficient mice are resistant to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced toxicity.

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Association of Shc, Cbl, Grb2, and Sos following treatment with 2,3,7,8-tetrachlorodibenzo-p-dioxin in primary rat hepatocytes

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TCDD (2,3,7,8-tetrachlorodibenzo-p-dioxin) causes an increase in protein tyrosine kinase activities at an early stage of poisoning in vivo in rat hepatocyte membranes

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Identification of c-Src as the integral component of the cytosolic Ah receptor complex, transducing the signal of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) through the protein phosphorylation pathway

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Treatment of rats during pubertal development with 2,3,7,8-tetrachlorodibenzo-p-dioxin alters both signaling kinase activities and epidermal growth factor receptor binding in the testis and the motility and acrosomal reaction of sperm

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The Ah receptor is not involved in 2,3,7,8-tetrachlorodibenzo- p-dioxin-mediated apoptosis in human leukemic T cell lines

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Transcriptional regulation by MAP kinases

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AP-1 function and regulation

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Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1

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2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD)-induced changes in activities of nuclear protein kinases and phosphatases affecting DNA binding activity of c-Myc and AP-1 in the livers of guinea pigs

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AP-1 and STAT mediate hyperoxia-induced gene transcription of heme oxygenase-1.

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Dioxin-dependent activation of murine Cyp1a-1 gene transcription requires protein kinase C-dependent phosphorylation

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Regulation of DNA binding activity of the ligand-activated aryl hydrocarbon receptor by tyrosine phosphorylation

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Constitutive activation of the aromatic hydrocarbon receptor

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The transcription factor GATA4 is activated by extracellular signal-regulated kinase 1- and 2-mediated phosphorylation of serine 105 in cardiomyocytes

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Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

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Aromatic hydrocarbon receptor interaction with the retinoblastoma protein potentiates repression of E2F-dependent transcription and cell cycle arrest

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A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells

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The Ah regulatory gene product. Survey of nineteen polycyclic aromatic compounds' and fifteen benzo[a]pyrene metabolites' capacity to bind to the cytosolic receptor

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Identification of a novel inhibitor of mitogen-activated protein kinase kinase

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Specific inhibitors of p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways block inducible nitric oxide synthase and tumor necrosis factor accumulation in murine macrophages stimulated with lipopolysaccharide and

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Is CYP1A1 induction always related to AHR signaling pathway?

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Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

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Molecular glue: kinase anchoring and scaffold proteins

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Activation of the estrogen receptor through phosphorylation by mitogen-activated protein kinase

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Estrogen receptor-mediated cross-talk with growth factor signaling pathways

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Inhibitors of serine/threonine-specific protein phosphatases stimulate transcription by the Ah receptor/Arnt dimer by affecting a step subsequent to XRE binding

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TCDD-mediated changes in hepatic epidermal growth factor receptor may be a critical event in the hepatocarcinogenic action of TCDD.

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Ultraviolet light and osmotic stress: activation of the JNK cascade through multiple growth factor and cytokine receptors

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Sustained increase in intracellular free calcium and activation of cyclooxygenase-2 expression in mouse hepatoma cells treated with dioxin

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CAMs and axonal growth: a critical evaluation of the role of calcium and the MAPK cascade

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Factors influencing elevation of intracellular Ca2+ in the MCF-10A human mammary epithelial cell line by carcinogenic polycyclic aromatic hydrocarbons

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Suppression of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-mediated CYP1A1 and CYP1B1 induction by 12-O-tetradecanoylphorbol-13-acetate: role of transforming growth factor beta and mitogen-activated protein kinases

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Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells

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2,3,7,8-Tetrachlorodibenzo-p-dioxin causes an increase in protein kinases associated with epidermal growth factor receptor in the hepatic plasma membrane

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PD98059 is an equipotent antagonist of the aryl hydrocarbon receptor and inhibitor of mitogen-activated protein kinase kinase

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10.1016/S0006-2952(02)01138-3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

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