(Q44137050)

30 November 2017

title

Neuronal overexpression of "readthrough" acetylcholinesterase is associated with antisense-suppressible behavioral impairments (English)

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30 November 2017

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Cohen O

1

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30 November 2017

Erb C

2

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30 November 2017

Ginzberg D

3

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30 November 2017

Pollak Y

4

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30 November 2017

Seidman S

5

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30 November 2017

Shoham S

6

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30 November 2017

Yirmiya R

7

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30 November 2017

Soreq H

8

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30 November 2017

language of work or name

English

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publication date

1 January 2002

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30 November 2017

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30 November 2017

volume

7

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30 November 2017

issue

8

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30 November 2017

page(s)

874-885

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https://scigraph.springernature.com/pub.10.1038/sj.mp.4001103

30 November 2017

exact match

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Acute stress facilitates long-lasting changes in cholinergic gene expression

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Control of alternative splicing of potassium channels by stress hormones.

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Tracking cholinergic pathways from psychological and chemical stressors to variable neurodeterioration paradigms.

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Structural roles of acetylcholinesterase variants in biology and pathology

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Excess "read-through" acetylcholinesterase attenuates but the "synaptic" variant intensifies neurodeterioration correlates

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Transgenic expression of human acetylcholinesterase induces progressive cognitive deterioration in mice.

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Enhanced hemicholinium binding and attenuated dendrite branching in cognitively impaired acetylcholinesterase-transgenic mice.

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Compensatory mechanisms enhance hippocampal acetylcholine release in transgenic mice expressing human acetylcholinesterase

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A transcription-activating polymorphism in the ACHE promoter associated with acute sensitivity to anti-acetylcholinesterases.

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Effect of reversed light-dark cycle on skin conductance in male rats.

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Antisense prevention of neuronal damages following head injury in mice

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Intracerebral administration of Mycoplasma fermentans produces sickness behavior: role of prostaglandins.

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Acetylcholinesterase enhances neurite growth and synapse development through alternative contributions of its hydrolytic capacity, core protein, and variable C termini

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Astrocyte hypertrophy in the dentate gyrus of young male rats reflects variation of individual stress rather than group environmental complexity manipulations

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Stress and cognitive function.

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Time course for synchronization of spontaneous locomotor activity in the rat following reversal of the daylight (12:12 h) cycle.

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Cholinergic modulation of a decrement in social investigation following repeated contacts between mice

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Ethological study of the effects of tetrahydroaminoacridine (THA) on social recognition in rats.

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Acetylcholinesterase-transgenic mice display embryonic modulations in spinal cord choline acetyltransferase and neurexin Ibeta gene expression followed by late-onset neuromotor deterioration

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Effects of mental stress on cardiac and motor rhythms.

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Specific modulation of social memory in rats by cholinomimetic and nootropic drugs, by benzodiazepine inverse agonists, but not by psychostimulants.

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Androgen-dependent vasopressinergic neurons are involved in social recognition in rats.

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Short duration of retroactive facilitation of social recognition in rats

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Acetylcholinesterase--new roles for an old actor

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Differential regulation of hippocampal glucocorticoid receptors mRNA and fast feedback: relevance to post-traumatic stress disorder.

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Learning/memory processes under stress conditions.

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Cortisol secretion and Alzheimer's disease progression.

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Oral administration of a corticotropin-releasing hormone receptor antagonist significantly attenuates behavioral, neuroendocrine, and autonomic responses to stress in primates

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7 January 2021

Psychological stress impairs spatial working memory: relevance to electrophysiological studies of hippocampal function.

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7 January 2021

Stress and glucocorticoids impair retrieval of long-term spatial memory

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Regional differences in the regulation of dopamine and noradrenaline release in medial frontal cortex, nucleus accumbens and caudate-putamen: a microdialysis study in the rat.

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Stress, depression and the mesolimbic dopamine system.

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7 January 2021

Stress Selectively Increases Fos Protein in Dopamine Neurons Innervating the Prefrontal Cortex

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7 January 2021

Neonatal excitotoxic ventral hippocampal damage alters dopamine response to mild repeated stress and to chronic haloperidol.

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7 January 2021

Anticholinesterases induce multigenic transcriptional feedback response suppressing cholinergic neurotransmission

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7 January 2021

Alzheimer's Disease: A Disorder of Cortical Cholinergic Innervation

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7 January 2021

Antisense technologies have a future fighting neurodegenerative diseases.

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7 January 2021

Complex host cell responses to antisense suppression of ACHE gene expression

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https://api.crossref.org/works/10.1038%2FSJ.MP.4001103

7 January 2021

Identifiers

DOI

10.1038/SJ.MP.4001103

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30 November 2017

1 reference