(Q44185566)

30 November 2017

title

Matrix remodeling in experimental and human heart failure: a possible regulatory role for TIMP-3. (English)

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30 November 2017

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Paul W M Fedak

1

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30 November 2017

Svetlana M Altamentova

2

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30 November 2017

Richard D Weisel

3

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30 November 2017

Nafiseh Nili

4

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30 November 2017

Nobuhisa Ohno

5

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30 November 2017

Subodh Verma

6

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30 November 2017

Tsu-Yee J Lee

7

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30 November 2017

Chris Kiani

8

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30 November 2017

Donald A G Mickle

9

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30 November 2017

Bradley H Strauss

10

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30 November 2017

Ren-Ke Li

11

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30 November 2017

publication date

10 October 2002

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American Journal of Physiology Heart and Circulatory Physiology

30 November 2017

published in

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30 November 2017

volume

284

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30 November 2017

issue

2

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30 November 2017

page(s)

H626-34

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Gene encoding a novel murine tissue inhibitor of metalloproteinases (TIMP), TIMP-3, is expressed in developing mouse epithelia, cartilage, and muscle, and is located on mouse chromosome 10.

30 November 2017

cites work

1 reference

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Doxycycline modulates smooth muscle cell growth, migration, and matrix remodeling after arterial injury

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A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells

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Tissue inhibitor of metalloproteinase-3 induces a Fas-associated death domain-dependent type II apoptotic pathway

21 January 2018

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Tumor necrosis factor-alpha and myocardial remodeling in progression of heart failure: a current perspective

21 January 2018

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Disruption of the sarcoglycan-sarcospan complex in vascular smooth muscle: a novel mechanism for cardiomyopathy and muscular dystrophy

21 January 2018

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Accelerated apoptosis in the Timp-3-deficient mammary gland

21 January 2018

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Downregulation of matrix metalloproteinases and reduction in collagen damage in the failing human heart after support with left ventricular assist devices

21 January 2018

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Proinflammatory cytokines regulate tissue inhibitors of metalloproteinases and disintegrin metalloproteinase in cardiac cells

21 January 2018

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ECM signalling: orchestrating cell behaviour and misbehaviour.

21 January 2018

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Extracellular matrix components influence the survival of adult cardiac myocytes in vitro.

21 January 2018

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Mechanisms and models in heart failure: A combinatorial approach

21 January 2018

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21 January 2018

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Identification of the Syrian hamster cardiomyopathy gene.

21 January 2018

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Effect of captopril on mortality and morbidity in patients with left ventricular dysfunction after myocardial infarction. Results of the survival and ventricular enlargement trial. The SAVE Investigators

21 January 2018

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Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, delta-sarcoglycan, in hamster: an animal model of disrupted dystrophin-associated glycoprotein complex

21 January 2018

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Matrix metalloproteinases: regulation and dysregulation in the failing heart

21 January 2018

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Myocardial matrix degradation and metalloproteinase activation in the failing heart: a potential therapeutic target

21 January 2018

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How matrix metalloproteinases regulate cell behavior

21 January 2018

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Mutations in the human delta-sarcoglycan gene in familial and sporadic dilated cardiomyopathy

21 January 2018

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Differential gene expression of extracellular matrix components in dilated cardiomyopathy.

21 January 2018

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The soluble catalytic domain of membrane type 1 matrix metalloproteinase cleaves the propeptide of progelatinase A and initiates autoproteolytic activation. Regulation by TIMP-2 and TIMP-3.

21 January 2018

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That impish TIMP: the tissue inhibitor of metalloproteinases-3

21 January 2018

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Reduction in myocardial collagen cross-linking parallels left ventricular dilatation in rat models of systolic chamber dysfunction

21 January 2018

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Cardiac Collagen Remodeling in the Cardiomyopathic Syrian Hamster and the Effect of Losartan

21 January 2018

1 reference

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10.1152/AJPHEART.00684.2002

21 January 2018

Identifiers

DOI

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30 November 2017

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