(Q44356848)

2 December 2017

title

Beta3 integrin deficiency promotes cardiac hypertrophy and inflammation (English)

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2 December 2017

Jie Ren

1

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2 December 2017

Joan Avery

2

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2 December 2017

Haibo Zhao

3

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2 December 2017

Jochen G Schneider

4

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2 December 2017

F Patrick Ross

5

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2 December 2017

Anthony J Muslin

6

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2 December 2017

language of work or name

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20 December 2006

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Journal of Molecular and Cellular Cardiology

2 December 2017

published in

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2 December 2017

volume

42

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2 December 2017

issue

2

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2 December 2017

page(s)

367-377

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The cellular and molecular response of cardiac myocytes to mechanical stress.

2 December 2017

cites work

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Role of integrins in cellular responses to mechanical stress and adhesion

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Mechanotransduction in response to shear stress. Roles of receptor tyrosine kinases, integrins, and Shc.

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Stretch of β1 Integrin Activates an Outwardly Rectifying Chloride Current via FAK and Src in Rabbit Ventricular Myocytes

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Integrins play a critical role in mechanical stress-induced p38 MAPK activation

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Integrin-mediated signal transduction linked to Ras pathway by GRB2 binding to focal adhesion kinase

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Integrin-mediated cell adhesion promotes tyrosine phosphorylation of p130Cas, a Src homology 3-containing molecule having multiple Src homology 2-binding motifs

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Cardiac myocyte-specific excision of the beta1 integrin gene results in myocardial fibrosis and cardiac failure

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RGD-containing peptides activate S6K1 through beta3 integrin in adult cardiac muscle cells

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Association of interleukin-6 -174G/C promoter polymorphism with hypertension and left ventricular hypertrophy in dialysis patients

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Dilated cardiomyopathy in transgenic mice with cardiac-specific overexpression of tumor necrosis factor-alpha

7 January 2021

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A third-generation lentivirus vector with a conditional packaging system

7 January 2021

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/J.YJMCC.2006.11.002

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2 December 2017

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