(Q44362424)

2 December 2017

title

AMPKα2 deficiency uncovers time dependency in the regulation of contraction-induced palmitate and glucose uptake in mouse muscle (English)

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2 December 2017

Marcia J Abbott

1

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2 December 2017

Lindsey D Bogachus

series ordinal

2

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2 December 2017

Lorraine P Turcotte

series ordinal

3

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2 December 2017

language of work or name

English

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publication date

5 May 2011

1 reference

Journal of Applied Physiology

2 December 2017

published in

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2 December 2017

volume

111

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2 December 2017

issue

1

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2 December 2017

page(s)

125-134

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CaMKK is an upstream signal of AMP-activated protein kinase in regulation of substrate metabolism in contracting skeletal muscle

2 December 2017

cites work

1 reference

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21 January 2018

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21 January 2018

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Skeletal muscle glucose uptake during contraction is regulated by nitric oxide and ROS independently of AMPK.

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Hormone-sensitive lipase activity and triacylglycerol hydrolysis are decreased in rat soleus muscle by cyclopiazonic acid

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Ca2+ and AMPK both mediate stimulation of glucose transport by muscle contractions

21 January 2018

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AMP kinase is required for mitochondrial biogenesis in skeletal muscle in response to chronic energy deprivation

21 January 2018

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Caffeine-induced Ca2+ release increases AMPK-dependent glucose uptake in rodent soleus muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.00807.2010

10.1152/JAPPLPHYSIOL.00807.2010

21 January 2018

Identifiers

DOI

1 reference

2 December 2017

1 reference