(Q44410474)

English

Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury

scientific article published on May 9, 2003

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scholarly article

1 reference

Europe PubMed Central

2 December 2017

Protein kinase C activation generates superoxide and contributes to impairment of cerebrovasodilation induced by G protein activation after brain injury (English)

1 reference

Europe PubMed Central

2 December 2017

author name string

William M. Armstead

1

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A12706231

26 December 2023

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publication date

1 May 2003

1 reference

Europe PubMed Central

2 December 2017

9 May 2003

1 reference

National Center for Biotechnology Information

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=12706231

26 December 2023

full work available at URL

https://api.elsevier.com/content/article/PII:S000689930302287X?httpAccept=text/xml

1 reference

10.1016/S0006-8993(03)02287-X

https://api.crossref.org/works/10.1016/S0006-8993(03)02287-X

26 December 2023

https://api.elsevier.com/content/article/PII:S000689930302287X?httpAccept=text/plain

1 reference

10.1016/S0006-8993(03)02287-X

https://api.crossref.org/works/10.1016/S0006-8993(03)02287-X

26 December 2023

1 reference

Europe PubMed Central

2 December 2017

971

1 reference

Europe PubMed Central

2 December 2017

2

1 reference

Europe PubMed Central

2 December 2017

153-160

1 reference

Europe PubMed Central

2 December 2017

G protein activation elicits cerebrovasodilation through interaction with K(ATP) and K(Ca) channels

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Superoxide generation links protein kinase C activation to impaired ATP-sensitive K+ channel function after brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Hypotension dilates pial arteries by KATP and kca channel activation.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Brain injury impairs ATP-sensitive K+ channel function in piglet cerebral arteries.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Different cerebral hemodynamic responses following fluid percussion brain injury in the newborn and juvenile pig.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Regulation of the cerebral circulation: role of endothelium and potassium channels

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Animate models of human head injury

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Liposome-entrapped superoxide dismutase reduces cerebral infarction in cerebral ischemia in rats

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Role of potassium channels in cerebral blood vessels.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Mastoparan may activate GTP hydrolysis by Gi-proteins in HL-60 membranes indirectly through interaction with nucleoside diphosphate kinase

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Activation of GTP formation and high-affinity GTP hydrolysis by mastoparan in various cell membranes. G-protein activation via nucleoside diphosphate kinase, a possible general mechanism of mastoparan action

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Mechanisms of coronary microvascular dilation induced by the activation of pertussis toxin-sensitive G proteins are vessel-size dependent. Heterogeneous involvement of nitric oxide pathway and ATP-sensitive K+ channels

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

The heterotrimeric G-protein Gi is localized to the insulin secretory granules of beta-cells and is involved in insulin exocytosis

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Superoxide production in experimental brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Ischemia alters cerebral vascular responses to hypercapnia and acetylcholine in piglets

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Postischemic cerebral microvascular responses to norepinephrine and hypotension in newborn pigs

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Swelling and blood flow in the injured child's brain

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Diversity of G proteins in signal transduction

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Influence of polyethylene glycol superoxide dismutase/catalase on altered opioid-induced pial artery dilation after brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Functional, morphological, and metabolic abnormalities of the cerebral microcirculation after concussive brain injury in cats

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2803%2902287-X

7 January 2021

Influence of brain injury on opioid-induced pial artery vasodilation

27

1 reference

10.1016/S0006-8993(03)02287-X

https://api.crossref.org/works/10.1016/S0006-8993(03)02287-X

26 December 2023

Identifiers

10.1016/S0006-8993(03)02287-X

1 reference

Europe PubMed Central

2 December 2017

1 reference

Europe PubMed Central

2 December 2017

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