(Q44419390)

2 December 2017

Bfl-1S, a novel alternative splice variant of Bfl-1, localizes in the nucleus via its C-terminus and prevents cell death (English)

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2 December 2017

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8

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2 December 2017

Jae-Kyun Ko

1

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2 December 2017

Min-Jung Lee

2

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2 December 2017

Sun-Hee Cho

3

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2 December 2017

Jung-Ah Cho

4

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2 December 2017

Bo-Young Lee

5

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2 December 2017

Jason Soonju Koh

6

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2 December 2017

Seung-Sook Lee

7

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2 December 2017

Chul-Woo Kim

9

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2 December 2017

language of work or name

English

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publication date

1 April 2003

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2 December 2017

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2 December 2017

22

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2 December 2017

2457-2465

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2 December 2017

16

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1206274

2 December 2017

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cites work

BCL-2 family: regulators of cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

The tumor suppressor ING1: structure and function

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

A1 functions at the mitochondria to delay endothelial apoptosis in response to tumor necrosis factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

NF-kappaB-mediated up-regulation of Bcl-x and Bfl-1/A1 is required for CD40 survival signaling in B lymphocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

Structural basis of BFL-1 for its interaction with BAX and its anti-apoptotic action in mammalian and yeast cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Upregulation of Bcl-x and Bfl-1 as a potential mechanism of chemoresistance, which can be overcome by NF-kappaB inhibition

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Functional dissection of Bfl-1, a Bcl-2 homolog: anti-apoptosis, oncogene-cooperation and cell proliferation activities

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

E1B 19,000-molecular-weight protein interacts with and inhibits CED-4-dependent, FLICE-mediated apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Cif (Cytochrome c efflux-inducing factor) activity is regulated by Bcl-2 and caspases and correlates with the activation of Bid.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Multiple gene duplication and expression of mouse bcl-2-related genes, A1.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Survival activity of Bcl-2 homologs Bcl-w and A1 only partially correlates with their ability to bind pro-apoptotic family members

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Expression of Bfl-1 in normal and tumor tissues: Bfl-1 overexpression in cancer is attributable to its preferential expression in infiltrating inflammatory cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Endothelial cell death induced by tumor necrosis factor-alpha is inhibited by the Bcl-2 family member, A1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Btf, a novel death-promoting transcriptional repressor that interacts with Bcl-2-related proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Cyclic re-entry of germinal center B cells and the efficiency of affinity maturation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Role of the C-terminal domain of Bax and Bcl-xL in their localization and function in yeast cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

The E1B 19K protein associates with lamins in vivo and its proper localization is required for inhibition of apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Bcl-2 family proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Transient expression of the Bcl-2 family member, A1-a, results in nuclear localization and resistance to staurosporine-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206274

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1206274

1 reference

2 December 2017

1 reference