(Q44538522)

3 December 2017

title

Temporal and spatial distribution of activated caspase-3 after subdural kainic acid infusions in rat spinal cord (English)

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3 December 2017

author name string

Stephanie A Nottingham

series ordinal

1

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3 December 2017

Joe E Springer

series ordinal

2

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3 December 2017

language of work or name

English

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publication date

1 September 2003

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The Journal of Comparative Neurology

3 December 2017

published in

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3 December 2017

volume

464

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3 December 2017

issue

4

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3 December 2017

page(s)

463-471

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Ca(2+) influx through AMPA or kainate receptors alone is sufficient to initiate excitotoxicity in cultured oligodendrocytes.

3 December 2017

cites work

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

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JNK3 contributes to c-jun induction and apoptosis in 4-hydroxynonenal-treated sympathetic neurons

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Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane

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AMPA-selective glutamate receptor subunits in glial cells of the adult bovine white matter.

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Temporal–Spatial Pattern of Acute Neuronal and Glial Loss after Spinal Cord Contusion

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Apoptosis of oligodendrocytes occurs for long distances away from the primary injury after compression trauma to rat spinal cord

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Mastoparan-induced apoptosis of cultured cerebellar granule neurons is initiated by calcium release from intracellular stores.

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Calcium, free radicals and excitotoxins in neuronal apoptosis.

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Oligodendrocytes from forebrain are highly vulnerable to AMPA/kainate receptor-mediated excitotoxicity.

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Alterations of Extracellular Calcium Elicit Selective Modes of Cell Death and Protease Activation in SH-SY5Y Human Neuroblastoma Cells

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Neurotoxic effects of excitatory amino acids in the mouse spinal cord: quisqualate and kainate but not N-methyl-D-aspartate induce permanent neural damage.

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7 January 2021

The role of calpain in caspase activation during etoposide induced apoptosis in T cells

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7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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7 January 2021

Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD

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7 January 2021

Delayed glial cell death following wallerian degeneration in white matter tracts after spinal cord dorsal column cordotomy in adult rats.

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7 January 2021

Calpain activation is upstream of caspases in radiation-induced apoptosis.

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7 January 2021

Dose-dependent reduction of tissue loss and functional impairment after spinal cord trauma with the AMPA/kainate antagonist NBQX

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7 January 2021

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7 January 2021

Delayed antagonism of AMPA/kainate receptors reduces long-term functional deficits resulting from spinal cord trauma

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7 January 2021

Alteration in the immunoreactivity of the calcineurin subunits after ischemic hippocampal damage

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7 January 2021

Neuronal apoptosis after CNS injury: the roles of glutamate and calcium.

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7 January 2021

Identifiers

DOI

10.1002/CNE.10806

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3 December 2017

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