(Q44583630)

4 December 2017

title

Genetic basis of kainate-induced excitotoxicity in mice: phenotypic modulation of seizure-induced cell death (English)

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4 December 2017

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Paula Elyse Schauwecker

series ordinal

1

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4 December 2017

publication date

1 August 2003

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4 December 2017

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4 December 2017

volume

55

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4 December 2017

issue

3

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4 December 2017

page(s)

201-210

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Kainate, a double agent that generates seizures: two decades of progress

4 December 2017

cites work

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7 January 2021

Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsy

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Kainic acid seizure syndrome and binding sites in developing rats.

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Ischemia-induced neuronal apoptosis

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7 January 2021

Glutamate neurotoxicity and diseases of the nervous system

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Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage

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7 January 2021

The role of excitotoxicity in neurodegenerative disease: implications for therapy

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7 January 2021

Differential susceptibility to seizures induced by systemic kainic acid treatment in mature DBA/2J and C57BL/6J mice

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7 January 2021

Mapping murine loci for seizure response to kainic acid

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Two methods for selective silver impregnation of degenerating axons and their synaptic endings in the central nervous system.

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Neuroprotective effect of ketamine administered after status epilepticus onset.

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The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage

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7 January 2021

Selective excitotoxic pathology in the rat hippocampus

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7 January 2021

Excitatory amino acids as a final common pathway for neurologic disorders.

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7 January 2021

Excitatory amino acid neurotoxicity and neurodegenerative disease

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7 January 2021

Kainic acid neurotoxicity toward hippocampal formation: dependence on specific excitatory pathways

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7 January 2021

25 Use of excitatory amino acids to make axon-sparing lesions of hypothalamus

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7 January 2021

Loss and reacquisition of hippocampal synapses after selective destruction of CA3-CA4 afferents with kainic acid

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7 January 2021

Microsatellite DNA variants between the FVB/N and C3HeB/FeJLe and C57BL/6J mouse strains

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7 January 2021

Maturation of kainic acid seizure-brain damage syndrome in the rat. II. Histopathological sequelae.

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7 January 2021

Excitatory amino acids and neuropsychiatric disorders

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7 January 2021

The role of specific ions in glutamate neurotoxicity.

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Resolution of quantitative traits into Mendelian factors by using a complete linkage map of restriction fragment length polymorphisms

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7 January 2021

Modification of seizure activity by electrical stimulation. II. Motor seizure

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7 January 2021

Excitotoxins: possible mechanisms of action

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7 January 2021

Genetic dissection of the signals that induce synaptic reorganization

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7 January 2021

Seizure-induced neuronal death is associated with induction of c-Jun N-terminal kinase and is dependent on genetic background.

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7 January 2021

Complications associated with genetic background effects in models of experimental epilepsy

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7 January 2021

Modulation of cell death by mouse genotype: differential vulnerability to excitatory amino acid-induced lesions

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7 January 2021

Differences in ionotropic glutamate receptor subunit expression are not responsible for strain-dependent susceptibility to excitotoxin-induced injury

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7 January 2021

Genetic determinants of susceptibility to excitotoxic cell death: implications for gene targeting approaches

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7 January 2021

Fluoro-Jade B: a high affinity fluorescent marker for the localization of neuronal degeneration

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7 January 2021

Kainic acid induced seizures: neurochemical and histopathological changes

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https://api.crossref.org/works/10.1016%2FS0920-1211%2803%2900115-3

7 January 2021

Kainic acid seizures in the developing brain: status epilepticus and spontaneous recurrent seizures.

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https://api.crossref.org/works/10.1016%2FS0920-1211%2803%2900115-3

7 January 2021

FVB/N: an inbred mouse strain preferable for transgenic analyses

1 reference

https://api.crossref.org/works/10.1016%2FS0920-1211%2803%2900115-3

7 January 2021

Maturation of kainic acid seizure-brain damage syndrome in the rat. I. Clinical, electrographic and metabolic observations.

1 reference

https://api.crossref.org/works/10.1016%2FS0920-1211%2803%2900115-3

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0920-1211%2803%2900115-3

7 January 2021

10.1016/S0920-1211(03)00115-3

Identifiers

DOI

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4 December 2017

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