(Q44664406)

English

Modulation of the reactive oxygen species (ROS) generation mediated by cyclic AMP-elevating agents or Interleukin 10 in granulocytes from type 2 diabetic patients (NIDDM): a PKA-independent phenomenon.

scientific article published in November 2003

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21 December 2019

Modulation of the reactive oxygen species (ROS) generation mediated by cyclic AMP-elevating agents or Interleukin 10 in granulocytes from type 2 diabetic patients (NIDDM): a PKA-independent phenomenon (English)

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Europe PubMed Central

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21 December 2019

reactive oxygen species

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Daniela Caldeira Costa

4

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21 December 2019

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J A Nogueira-Machado

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21 December 2019

F C Lima e Silva

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21 December 2019

L O Medina

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21 December 2019

M M Chaves

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21 December 2019

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1 November 2003

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21 December 2019

Diabetes and Metabolism

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21 December 2019

29

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21 December 2019

5

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21 December 2019

533-537

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21 December 2019

Hyperglycemia and insulin resistance: possible mechanisms

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Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

1 reference

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Effect in vitro of gamma interferon and interleukin-10 on generation of oxidizing species by human granulocytes

1 reference

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A simple technique for simultaneous human leukocytes separation

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Nitric oxide and oxygen radicals: a question of balance

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Peroxynitrite: a biologically significant oxidant

1 reference

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7 January 2021

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Oxidants, antioxidants, and the degenerative diseases of aging

1 reference

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Anti-inflammatory activities of cAMP-elevating agents: enhancement of IL-10 synthesis and concurrent suppression of TNF production.

1 reference

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Differential signaling of cyclic AMP: opposing effects of exchange protein directly activated by cyclic AMP and cAMP-dependent protein kinase on protein kinase B activation

1 reference

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PKA phosphorylation of Src mediates cAMP's inhibition of cell growth via Rap1.

1 reference

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G protein pathways

1 reference

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7 January 2021

based on heuristic

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Identifiers

10.1016/S1262-3636(07)70068-X

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Europe PubMed Central

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21 December 2019

PubMed publication ID

1 reference

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21 December 2019

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