(Q44682493)

5 December 2017

title

Cannabinoid CB1 receptor activation does not prevent the toxicity of glutamate towards embryonic chick telencephalon primary cultures (English)

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5 December 2017

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Olov Nilsson

1

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5 December 2017

Stig O P Jacobsson

series ordinal

2

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5 December 2017

Christopher J Fowler

series ordinal

3

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5 December 2017

language of work or name

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1 November 2003

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Comparative Biochemistry and Physiology C: Toxicology and Pharmacology

5 December 2017

published in

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5 December 2017

volume

136

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5 December 2017

issue

3

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5 December 2017

page(s)

245-251

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Neurotoxicity of glutamate in chick telencephalon neurons: reduction of toxicity by preincubation with carbachol, but not by the endogenous fatty acid amides anandamide and palmitoylethanolamide

5 December 2017

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Mechanisms of apoptosis in embryonic cortical neurons (E6 and E7) in culture involve lipid signalling, protein phosphorylation and caspase activation

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Differences in the pharmacological properties of rat and chicken brain fatty acid amidohydrolase

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Pharmacological properties of cannabinoid receptors in the avian brain: similarity of rat and chicken cannabinoid1 receptor recognition sites and expression of cannabinoid2 receptor-like immunoreactivity in the embryonic chick brain

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Cannabinoid receptor activation and elevated cyclic AMP reduce glutamate neurotoxicity.

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Blockade of cannabinoid CB(1) receptor function protects against in vivo disseminating brain damage following NMDA-induced excitotoxicity.

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Anandamide, but not 2-arachidonoylglycerol, accumulates during in vivo neurodegeneration

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Dopamine and glutamate neurotoxicity in cultured chick telencephali cells: effects of NMDA antagonists, antioxidants and MAO inhibitors

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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Identifiers

DOI

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5 December 2017

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