(Q44774249)

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Differential signalling mechanisms predisposing primary human skeletal muscle cells to altered proliferation and differentiation: roles of IGF-I and TNFalpha

scientific article published in March 2004

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scholarly article

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Europe PubMed Central

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6 December 2017

Differential signalling mechanisms predisposing primary human skeletal muscle cells to altered proliferation and differentiation: roles of IGF-I and TNFalpha (English)

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Europe PubMed Central

PubMed publication ID

6 December 2017

skeletal muscle cell

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based on heuristic

inferred from title

Claire E. Stewart

5

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Europe PubMed Central

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6 December 2017

author name string

Emily J Foulstone

1

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6 December 2017

Camille Huser

2

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6 December 2017

Anna L Crown

3

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6 December 2017

Jeff M P Holly

4

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6 December 2017

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publication date

1 March 2004

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6 December 2017

Experimental Cell Research

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6 December 2017

294

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6 December 2017

1

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6 December 2017

223-235

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6 December 2017

Catabolic illness. Strategies for enhancing recovery

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Growth, differentiation, and survival: multiple physiological functions for insulin-like growth factors

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Phosphatidylinositol 3-kinase inhibitors block differentiation of skeletal muscle cells

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Myogenic differentiation requires signalling through both phosphatidylinositol 3-kinase and p38 MAP kinase

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The mitogenic and myogenic actions of insulin-like growth factors utilize distinct signaling pathways.

1 reference

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Characterisation of the IGF system in a primary adult human skeletal muscle cell model, and comparison of the effects of insulin and IGF-I on protein metabolism

1 reference

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7 January 2021

Role of insulin-like growth factor binding protein-3 (IGFBP-3) in the differentiation of primary human adult skeletal myoblasts

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Tumors secreting human TNF/cachectin induce cachexia in mice

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Long-term inhibition of tumor growth by tumor necrosis factor in the absence of cachexia or T-cell immunity

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Tumor necrosis factor-alpha-induced apoptosis is associated with suppression of insulin-like growth factor binding protein-5 secretion in differentiating murine skeletal myoblasts

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7 January 2021

Insulin-like growth factors (IGF-I and IGF-II) inhibit C2 skeletal myoblast differentiation and enhance TNF alpha-induced apoptosis

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Glycogen synthase activity is reduced in cultured skeletal muscle cells of non-insulin-dependent diabetes mellitus subjects. Biochemical and molecular mechanisms

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7 January 2021

The expression of TNF alpha by human muscle. Relationship to insulin resistance

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Electrical impedance in assessing human body composition: the BIA method

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Isolation and characterization of human muscle cells

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Tumor necrosis factor inhibits human myogenesis in vitro

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Initiation of satellite cell replication in bupivacaine-induced myonecrosis.

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Effects of tumor necrosis factor-alpha on insulin action in cultured human muscle cells

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Insulin-like growth factor I stimulates myoblast expansion and myofiber development in the limb

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7 January 2021

Myogenic vector expression of insulin-like growth factor I stimulates muscle cell differentiation and myofiber hypertrophy in transgenic mice

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IGF-I is required for normal embryonic growth in mice

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The MAPK signaling cascade

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Distinct signaling pathways regulate transformation and inhibition of skeletal muscle differentiation by oncogenic Ras

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Stress-activated protein kinase-2/p38 and a rapamycin-sensitive pathway are required for C2C12 myogenesis

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GSK3 takes centre stage more than 20 years after its discovery

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Insulin receptor substrate-1 and phosphatidylinositol 3-kinase regulate extracellular signal-regulated kinase-dependent and -independent signaling pathways during myogenic differentiation

1 reference

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7 January 2021

Mediation of IGF-1-induced skeletal myotube hypertrophy by PI(3)K/Akt/mTOR and PI(3)K/Akt/GSK3 pathways.

1 reference

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7 January 2021

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10.1016/J.YEXCR.2003.10.034

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

6 December 2017

ResearchGate publication ID

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