(Q44854919)

6 December 2017

Abnormalities of NBR1, a novel autophagy-associated protein, in muscle fibers of sporadic inclusion-body myositis (English)

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6 December 2017

1 reference

Carla D'Agostino

1

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6 December 2017

Anna Nogalska

2

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6 December 2017

Mafalda Cacciottolo

3

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6 December 2017

W King Engel

4

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6 December 2017

Valerie Askanas

5

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6 December 2017

21 September 2011

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6 December 2017

Acta Neuropathologica

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6 December 2017

122

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6 December 2017

5

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6 December 2017

627-636

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https://scigraph.springernature.com/pub.10.1007/s00401-011-0874-3

6 December 2017

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cites work

beta-Amyloid precursor epitopes in muscle fibers of inclusion body myositis

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Inclusion-body myositis: muscle-fiber molecular pathology and possible pathogenic significance of its similarity to Alzheimer's and Parkinson's disease brains

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Sporadic inclusion-body myositis: conformational multifactorial ageing-related degenerative muscle disease associated with proteasomal and lysosomal inhibition, endoplasmic reticulum stress, and accumulation of amyloid-β42 oligomers and phosphorylat

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Novel immunolocalization of alpha-synuclein in human muscle of inclusion-body myositis, regenerating and necrotic muscle fibers, and at neuromuscular junctions

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Inclusion body myositis: a degenerative muscle disease associated with intra-muscle fiber multi-protein aggregates, proteasome inhibition, endoplasmic reticulum stress and decreased lysosomal degradation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Amyloid peptides in different assembly states and related effects on isolated and cellular proteasomes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Inflammatory muscle diseases: a critical review on pathogenesis and therapies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Sorting, recognition and activation of the misfolded protein degradation pathways through macroautophagy and the proteasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Proteasome inhibition and aggresome formation in sporadic inclusion-body myositis and in amyloid-beta precursor protein-overexpressing cultured human muscle fibers

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Regulation mechanisms and signaling pathways of autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Alzheimer neurofibrillary degeneration: significance, etiopathogenesis, therapeutics and prevention

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

p62 Targeting to the autophagosome formation site requires self-oligomerization but not LC3 binding

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Selective autophagy mediated by autophagic adapter proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

NBR1 cooperates with p62 in selective autophagy of ubiquitinated targets.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

A role for NBR1 in autophagosomal degradation of ubiquitinated substrates

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Small assemblies of unmodified amyloid beta-protein are the proximate neurotoxin in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Early accumulation of p62 in neurofibrillary tangles in Alzheimer's disease: possible role in tangle formation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Intracellular amyloid-beta in Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

The kinase domain of titin controls muscle gene expression and protein turnover

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy is required to maintain muscle mass.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy inhibition induces atrophy and myopathy in adult skeletal muscles.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Epoxomicin, a potent and selective proteasome inhibitor, exhibits in vivo antiinflammatory activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy in neurodegenerative disease: friend, foe or turncoat?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy, amyloidogenesis and Alzheimer disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy failure in Alzheimer's disease--locating the primary defect

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Decreased SIRT1 deacetylase activity in sporadic inclusion-body myositis muscle fibers

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Novel demonstration of amyloid-β oligomers in sporadic inclusion-body myositis muscle fibers

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Impaired autophagy in sporadic inclusion-body myositis and in endoplasmic reticulum stress-provoked cultured human muscle fibers

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

p62/SQSTM1 is overexpressed and prominently accumulated in inclusions of sporadic inclusion-body myositis muscle fibers, and can help differentiating it from polymyositis and dermatomyositis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Autophagy in skeletal muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Sequestosome 1/p62 is a polyubiquitin chain binding protein involved in ubiquitin proteasome degradation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Bafilomycin A1 inhibits chloroquine-induced death of cerebellar granule neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

In AbetaPP-overexpressing cultured human muscle fibers proteasome inhibition enhances phosphorylation of AbetaPP751 and GSK3beta activation: effects mitigated by lithium and apparently relevant to sporadic inclusion-body myositis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Abeta inhibits the proteasome and enhances amyloid and tau accumulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Amyloid-beta42 is preferentially accumulated in muscle fibers of patients with sporadic inclusion-body myositis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Interactions with LC3 and polyubiquitin chains link nbr1 to autophagic protein turnover

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Conformational change as one of the earliest alterations of tau in Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

NOGO is increased and binds to BACE1 in sporadic inclusion-body myositis and in A beta PP-overexpressing cultured human muscle fibers.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

Sequential phosphorylation of Tau by glycogen synthase kinase-3beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

NBR1 and p62 as cargo receptors for selective autophagy of ubiquitinated targets

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00401-011-0874-3

10.1007/S00401-011-0874-3

21 January 2018

Identifiers

DOI

1 reference

6 December 2017

1 reference