(Q44929738)

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Repetitive transcranial magnetic stimulation increases the release of dopamine in the nucleus accumbens shell of morphine-sensitized rats during abstinence.

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29 December 2019

Repetitive transcranial magnetic stimulation increases the release of dopamine in the nucleus accumbens shell of morphine-sensitized rats during abstinence (English)

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29 December 2019

nucleus accumbens

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3

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29 December 2019

Nicolas Singewald

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29 December 2019

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A Erhardt

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29 December 2019

I Sillaber

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29 December 2019

M B Müller

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29 December 2019

M E Keck

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29 December 2019

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1 November 2004

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29 December 2019

Neuropsychopharmacology

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29 December 2019

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29 December 2019

11

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29 December 2019

2074-2080

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29 December 2019

https://scigraph.springernature.com/pub.10.1038/sj.npp.1300493

0 references

Depression of mesolimbic dopamine transmission and sensitization to morphine during opiate abstinence

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Reciprocal changes in dopamine responsiveness in the nucleus accumbens shell and core and in the dorsal caudate-putamen in rats sensitized to morphine.

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Lasting reduction in mesolimbic dopamine neuronal activity after morphine withdrawal.

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Preferential stimulation of dopamine release in the nucleus accumbens by opiates, alcohol, and barbiturates: studies with transcerebral dialysis in freely moving rats

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Effects of acute repetitive transcranial magnetic stimulation on dopamine release in the rat dorsolateral striatum

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Repetitive transcranial magnetic stimulation induces active coping strategies and attenuates the neuroendocrine stress response in rats.

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Repetitive transcranial magnetic stimulation increases the release of dopamine in the mesolimbic and mesostriatal system

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Neuroendocrine and behavioral effects of repetitive transcranial magnetic stimulation in a psychopathological animal model are suggestive of antidepressant-like effects

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Unilateral left prefrontal transcranial magnetic stimulation (TMS) produces intensity-dependent bilateral effects as measured by interleaved BOLD fMRI.

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Modulation of drug-induced sensitization processes by endogenous opioid systems

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Long lasting changes in morphine-induced mesolimbic dopamine release after chronic morphine exposure.

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The dopamine hypothesis of reward: past and current status

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A single exposure to morphine induces long-lasting behavioural and neurochemical sensitization in rats

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Alterations in dopaminergic and glutamatergic transmission in the induction and expression of behavioral sensitization: a critical review of preclinical studies

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Sensitization occurs to the locomotor effects of morphine and the specific mu opioid receptor agonist, DAGO, administered repeatedly to the ventral tegmental area but not to the nucleus accumbens

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Mu opioid receptor signaling in morphine sensitization

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Transcranial magnetic stimulation induces increases in extracellular levels of dopamine and glutamate in the nucleus accumbens

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