(Q44970310)

7 December 2017

title

Adenylyl cyclase type VI corrects cardiac sarcoplasmic reticulum calcium uptake defects in cardiomyopathy (English)

1 reference

7 December 2017

Tong Tang

1

1 reference

7 December 2017

Mei Hua Gao

2

1 reference

7 December 2017

David M Roth

3

1 reference

7 December 2017

Tracy Guo

4

1 reference

7 December 2017

H Kirk Hammond

5

1 reference

7 December 2017

publication date

8 July 2004

1 reference

American Journal of Physiology Heart and Circulatory Physiology

7 December 2017

published in

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7 December 2017

volume

287

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7 December 2017

issue

5

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7 December 2017

page(s)

H1906-12

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Dilated cardiomyopathy and sudden death resulting from constitutive activation of protein kinase a.

7 December 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse hearts

21 January 2018

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How do beta-blockers improve ventricular function in patients with congestive heart failure?

21 January 2018

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Cardiac excitation-contraction coupling

21 January 2018

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PKC-alpha regulates cardiac contractility and propensity toward heart failure

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Of phospholamban, mice, and humans with heart failure.

21 January 2018

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Transgenic Galphaq overexpression induces cardiac contractile failure in mice

21 January 2018

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Interactions between phospholamban and beta-adrenergic drive may lead to cardiomyopathy and early mortality

21 January 2018

1 reference

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Low- and high-level transgenic expression of beta2-adrenergic receptors differentially affect cardiac hypertrophy and function in Galphaq-overexpressing mice

21 January 2018

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Altered calcium handling is critically involved in the cardiotoxic effects of chronic beta-adrenergic stimulation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Increased expression of adenylylcyclase type VI proportionately increases beta-adrenergic receptor-stimulated production of cAMP in neonatal rat cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Protein Kinase Cα Negatively Regulates Systolic and Diastolic Function in Pathological Hypertrophy

21 January 2018

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Chronic suppression of heart-failure progression by a pseudophosphorylated mutant of phospholamban via in vivo cardiac rAAV gene delivery

21 January 2018

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Is depressed myocyte contractility centrally involved in heart failure?

21 January 2018

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Chronic phospholamban inhibition prevents progressive cardiac dysfunction and pathological remodeling after infarction in rats.

21 January 2018

1 reference

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Cardiac-specific overexpression of phospholamban alters calcium kinetics and resultant cardiomyocyte mechanics in transgenic mice

21 January 2018

1 reference

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Patients with end-stage congestive heart failure treated with beta-adrenergic receptor antagonists have improved ventricular myocyte calcium regulatory protein abundance

21 January 2018

1 reference

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Intracoronary adenovirus encoding adenylyl cyclase VI increases left ventricular function in heart failure

21 January 2018

1 reference

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What is the role of beta-adrenergic signaling in heart failure?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Targeted ablation of the phospholamban gene is associated with markedly enhanced myocardial contractility and loss of beta-agonist stimulation

21 January 2018

1 reference

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Phospholamban: a crucial regulator of cardiac contractility

21 January 2018

1 reference

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PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

21 January 2018

1 reference

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β1-adrenergic receptor polymorphisms confer differential function and predisposition to heart failure

21 January 2018

1 reference

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Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

The vmax of the Ca2+-ATPase of cardiac sarcoplasmic reticulum (SERCA2a) is not altered by Ca2+/calmodulin-dependent phosphorylation or by interaction with phospholamban

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Beta-blockers restore calcium release channel function and improve cardiac muscle performance in human heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Cardiac-directed adenylyl cyclase expression improves heart function in murine cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Adenylyl cyclase increases survival in cardiomyopathy

21 January 2018

1 reference

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Dilated cardiomyopathy and heart failure caused by a mutation in phospholamban

21 January 2018

1 reference

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Heart failure -- a challenge to our current concepts of excitation-contraction coupling

21 January 2018

1 reference

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Rescue of cardiomyocyte dysfunction by phospholamban ablation does not prevent ventricular failure in genetic hypertrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Complexity and diversity of mammalian adenylyl cyclases

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Phosphorylation by mitogen-activated protein kinase mediates the hypoxia-induced turnover of the TAL1/SCL transcription factor in endothelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

Cardiac-specific overexpression of Galphaq alters excitation-contraction coupling in isolated cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00356.2004

10.1152/AJPHEART.00356.2004

21 January 2018

Identifiers

DOI

1 reference

7 December 2017

1 reference