(Q45300283)

11 December 2017

title

Polyglutamine-induced ion channels: a possible mechanism for the neurotoxicity of Huntington and other CAG repeat diseases (English)

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11 December 2017

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Hirakura Y

1

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11 December 2017

Azimov R

series ordinal

2

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11 December 2017

Azimova R

series ordinal

3

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11 December 2017

Kagan BL

series ordinal

4

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11 December 2017

language of work or name

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1 May 2000

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Journal of Neuroscience Research

11 December 2017

published in

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11 December 2017

volume

60

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11 December 2017

issue

4

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11 December 2017

page(s)

490-494

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Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum

11 December 2017

cites work

1 reference

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The mechanism of islet amyloid polypeptide toxicity is membrane disruption by intermediate-sized toxic amyloid particles.

21 January 2018

1 reference

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Antimicrobial defensin peptides form voltage-dependent ion-permeable channels in planar lipid bilayer membranes

21 January 2018

1 reference

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Peptides containing glutamine repeats as substrates for transglutaminase-catalyzed cross-linking: relevance to diseases of the nervous system

21 January 2018

1 reference

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Synthetic mammalian C-type natriuretic peptide forms large cation channels

21 January 2018

1 reference

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Channel formation by a neurotoxic prion protein fragment

21 January 2018

1 reference

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Polyglutamines, nuclear inclusions and neurodegeneration

21 January 2018

1 reference

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Glutamine repeats as polar zippers: their possible role in inherited neurodegenerative diseases

21 January 2018

1 reference

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Cytochrome c: can't live with it--can't live without it.

21 January 2018

1 reference

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Amyloid beta protein-(1-42) forms calcium-permeable, Zn2+-sensitive channel

21 January 2018

1 reference

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Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo

21 January 2018

1 reference

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Hypoxic enhancement of quantal catecholamine secretion. Evidence for the involvement of amyloid beta-peptides.

21 January 2018

1 reference

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Neuronal intranuclear inclusions in polyglutamine diseases: nuclear weapons or nuclear fallout?

21 January 2018

1 reference

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Channel formation in planar lipid bilayers by a neurotoxic fragment of the beta-amyloid peptide

21 January 2018

1 reference

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Pore formation by the cytotoxic islet amyloid peptide amylin

21 January 2018

1 reference

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Use of planar lipid bilayer membranes for rapid screening of membrane active compounds

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2F%28SICI%291097-4547%2820000515%2960%3A4%3C490%3A%3AAID-JNR7%3E3.0.CO%3B2-9

10.1002/(SICI)1097-4547(20000515)60:4<490::AID-JNR7>3.0.CO;2-9

21 January 2018

Identifiers

DOI

1 reference

11 December 2017

1 reference