(Q45625417)

English

A non-consensus branch point plays an important role in determining the stability of the 2-kb LAT intron during acute and latent infections of herpes simplex virus type-1.

scientific article published in July 2004

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

A non-consensus branch point plays an important role in determining the stability of the 2-kb LAT intron during acute and latent infections of herpes simplex virus type-1. (English)

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

author name string

Ruma Mukerjee

1

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

Wen Kang

2

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

Vikram Suri

3

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

Nigel W Fraser

4

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

language of work or name

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publication date

1 July 2004

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

324

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

2

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

340-349

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

Regions of the herpes simplex virus type 1 latency-associated transcript that protect cells from apoptosis in vitro and protect neuronal cells in vivo

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

An HSV LAT null mutant reactivates slowly from latent infection and makes small plaques on CV-1 monolayers

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

ICP0, a regulator of herpes simplex virus during lytic and latent infection

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Herpes simplex virus latency-associated transcript is a stable intron

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

ICP0 is required for efficient reactivation of herpes simplex virus type 1 from neuronal latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Herpes simplex virus latent phase transcription facilitates in vivo reactivation

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Region of herpes simplex virus type 1 latency-associated transcript sufficient for wild-type spontaneous reactivation promotes cell survival in tissue culture

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Establishment and maintenance of HSV latent infection is mediated through correct splicing of the LAT primary transcript

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

The promoter of the latency-associated transcripts of herpes simplex virus type 1 contains a functional cAMP-response element: role of the latency-associated transcripts and cAMP in reactivation of viral latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Correlation between herpes simplex virus type 1 rate of reactivation from latent infection and the number of infected neurons in trigeminal ganglia

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Mapping of low abundance latency-associated RNA in the trigeminal ganglia of mice latently infected with herpes simplex virus type 1.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Virus-induced neuronal apoptosis blocked by the herpes simplex virus latency-associated transcript.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

The 2-kilobase intron of the herpes simplex virus type 1 latency-associated transcript has a half-life of approximately 24 hours in SY5Y and COS-1 cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Herpes simplex virus type 1 latency-associated transcript gene promotes neuronal survival

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Experimental investigation of herpes simplex virus latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Sequence of the latency-related gene of herpes simplex virus type 1.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

Molecular analysis of herpes simplex virus type 1 during epinephrine-induced reactivation of latently infected rabbits in vivo.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

Herpes simplex virus type 1 latency-associated transcripts suppress viral replication and reduce immediate-early gene mRNA levels in a neuronal cell line

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

The latency-associated transcript gene of herpes simplex virus type 1 (HSV-1) is required for efficient in vivo spontaneous reactivation of HSV-1 from latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

Expression of herpes simplex virus type 1 latency-associated transcripts in the trigeminal ganglia of mice during acute infection and reactivation of latent infection

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

The herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latency

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

The herpes simplex virus latency-associated transcript is spliced during the latent phase of infection

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

Evidence that two latency-associated transcripts of herpes simplex virus type 1 are nonlinear

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

The herpes simplex virus type 1 2.0-kilobase latency-associated transcript is a stable intron which branches at a guanosine

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.03.043

7 January 2021

based on heuristic

inferred from DOI database lookup

Identifiers

10.1016/J.VIROL.2004.03.043

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

release_klwfwh2trngi7ek3vzulogvlva

1 reference

https://api.fatcat.wiki/v0/release/klwfwh2trngi7ek3vzulogvlva

24 November 2022

based on heuristic

mapped directly with Wikidata item

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 December 2017

ResearchGate publication ID

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