(Q46094743)

19 December 2017

title

The transcription factor Eya2 prevents pressure overload-induced adverse cardiac remodeling (English)

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19 December 2017

author name string

Seung Hee Lee

1

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19 December 2017

Dong Kwon Yang

2

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19 December 2017

Bo Youn Choi

3

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19 December 2017

Young-Hoon Lee

4

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19 December 2017

Seon-Young Kim

5

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19 December 2017

Dongtak Jeong

6

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19 December 2017

Roger J Hajjar

7

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19 December 2017

Woo Jin Park

8

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19 December 2017

publication date

14 January 2009

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19 December 2017

Journal of Molecular and Cellular Cardiology

published in

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19 December 2017

volume

46

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19 December 2017

issue

4

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19 December 2017

page(s)

596-605

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19 December 2017

Phenotyping hypertrophy: eschew obfuscation

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Eyes absent mediates cross-talk between retinal determination genes and the receptor tyrosine kinase signaling pathway

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Mouse Eya genes are expressed during limb tendon development and encode a transcriptional activation function

7 January 2021

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Eyes absent represents a class of protein tyrosine phosphatases

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The transcription factor Eyes absent is a protein tyrosine phosphatase

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A comparative study of Eya1 and Eya4 protein function and its implication in branchio-oto-renal syndrome and DFNA10

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Mutation in the transcriptional coactivator EYA4 causes dilated cardiomyopathy and sensorineural hearing loss

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7 January 2021

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Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy

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Activation or inactivation of cardiac Akt/mTOR signaling diverges physiological from pathological hypertrophy

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Physiological myocardial hypertrophy: how and why?

7 January 2021

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NFATc2 is a necessary mediator of calcineurin-dependent cardiac hypertrophy and heart failure

7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Identifiers

DOI

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19 December 2017

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19 December 2017