(Q46158773)

20 December 2017

title

Smad signaling in the rat model of monocrotaline pulmonary hypertension (English)

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20 December 2017

author name string

Margaret F Ramos

1

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20 December 2017

Michael W Lamé

2

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20 December 2017

Henry J Segall

3

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20 December 2017

Dennis W Wilson

4

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20 December 2017

language of work or name

English

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publication date

1 February 2008

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20 December 2017

published in

Toxicologic Pathology

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20 December 2017

volume

36

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20 December 2017

issue

2

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20 December 2017

page(s)

311-320

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20 December 2017

Primary pulmonary hypertension is associated with reduced pulmonary vascular expression of type II bone morphogenetic protein receptor

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1 reference

https://api.crossref.org/works/10.1177%2F0192623307311402

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Familial primary pulmonary hypertension (gene PPH1) is caused by mutations in the bone morphogenetic protein receptor-II gene

21 January 2018

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Smad-dependent and Smad-independent pathways in TGF-beta family signalling

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Direct signaling by the BMP type II receptor via the cytoskeletal regulator LIMK1

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Balancing the activation state of the endothelium via two distinct TGF-beta type I receptors

21 January 2018

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Overexpression of Smad7 results in severe pathological alterations in multiple epithelial tissues

21 January 2018

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Nucleocytoplasmic shuttling of Smads 2, 3, and 4 permits sensing of TGF-beta receptor activity

21 January 2018

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Truncated type II receptor for BMP-4 induces secondary axial structures in Xenopus embryos

21 January 2018

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Protein targets of monocrotaline pyrrole in pulmonary artery endothelial cells

21 January 2018

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Monocrotaline pyrrole targets proteins with and without cysteine residues in the cytosol and membranes of human pulmonary artery endothelial cells

21 January 2018

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Heterozygous germline mutations in BMPR2, encoding a TGF-beta receptor, cause familial primary pulmonary hypertension

21 January 2018

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21 January 2018

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Mutations of the TGF-beta type II receptor BMPR2 in pulmonary arterial hypertension.

21 January 2018

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21 January 2018

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Functional interaction between BMPR-II and Tctex-1, a light chain of Dynein, is isoform-specific and disrupted by mutations underlying primary pulmonary hypertension

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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The BMP type II receptor is located in lipid rafts, including caveolae, of pulmonary endothelium in vivo and in vitro

21 January 2018

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Monocrotaline pyrrole induces Smad nuclear accumulation and altered signaling expression in human pulmonary arterial endothelial cells

21 January 2018

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Caveolin-1 regulates transforming growth factor (TGF)-beta/SMAD signaling through an interaction with the TGF-beta type I receptor

21 January 2018

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21 January 2018

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21 January 2018

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Caveolin-1 deficiency (-/-) conveys premalignant alterations in mammary epithelia, with abnormal lumen formation, growth factor independence, and cell invasiveness

21 January 2018

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Sporadic primary pulmonary hypertension is associated with germline mutations of the gene encoding BMPR-II, a receptor member of the TGF-beta family

21 January 2018

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21 January 2018

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Caveolin-1 in oncogenic transformation, cancer, and metastasis

21 January 2018

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Mechanisms and pathology of monocrotaline pulmonary toxicity

21 January 2018

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https://api.crossref.org/works/10.1177%2F0192623307311402

21 January 2018

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https://api.crossref.org/works/10.1177%2F0192623307311402

Nucleocytoplasmic shuttling of Smad1 conferred by its nuclear localization and nuclear export signals

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F0192623307311402

Dysfunctional Smad Signaling Contributes to Abnormal Smooth Muscle Cell Proliferation in Familial Pulmonary Arterial Hypertension

21 January 2018

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https://api.crossref.org/works/10.1177%2F0192623307311402

Microvascular regeneration in established pulmonary hypertension by angiogenic gene transfer.

21 January 2018

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https://api.crossref.org/works/10.1177%2F0192623307311402

Defects in caveolin-1 cause dilated cardiomyopathy and pulmonary hypertension in knockout mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F0192623307311402

Caveolin-1 null mice are viable but show evidence of hyperproliferative and vascular abnormalities

21 January 2018

1 reference

24 May 2022

https://opencitations.net/index/coci/api/v1/citations/10.1074/JBC.M105408200

Identifiers

DOI

10.1177/0192623307311402

1 reference

20 December 2017

1 reference

20 December 2017