(Q46182214)

20 December 2017

title

TIA nuclear proteins regulate the alternate splicing of lysyl hydroxylase 2. (English)

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20 December 2017

author name string

Heather N Yeowell

1

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20 December 2017

Linda C Walker

2

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20 December 2017

David M Mauger

3

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20 December 2017

Puneet Seth

4

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20 December 2017

Mariano A Garcia-Blanco

5

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20 December 2017

language of work or name

English

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publication date

25 December 2008

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20 December 2017

Journal of Investigative Dermatology

published in

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20 December 2017

volume

129

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20 December 2017

issue

6

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20 December 2017

page(s)

1402-1411

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20 December 2017

Fox-2 mediates epithelial cell-specific fibroblast growth factor receptor 2 exon choice

cites work

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7 January 2021

RNA-binding protein TIAR is essential for primordial germ cell development

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A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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Interleukin 4 and prolonged hypoxia induce a higher gene expression of lysyl hydroxylase 2 and an altered cross-link pattern: important pathogenetic steps in early and late stage of systemic scleroderma?

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

An intronic sequence element mediates both activation and repression of rat fibroblast growth factor receptor 2 pre-mRNA splicing

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7 January 2021

An intronic splicing silencer causes skipping of the IIIb exon of fibroblast growth factor receptor 2 through involvement of polypyrimidine tract binding protein

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Alternative splicing in disease and therapy

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

A class of human exons with predicted distant branch points revealed by analysis of AG dinucleotide exclusion zones

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7 January 2021

Using CLUSTAL for multiple sequence alignments

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Evidence for increased pyridinoline concentration in fibrotic tissues in diffuse systemic sclerosis

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Two isoforms of the T-cell intracellular antigen 1 (TIA-1) splicing factor display distinct splicing regulation activities. Control of TIA-1 isoform ratio by TIA-1-related protein

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

TIA-1 and TIAR activate splicing of alternative exons with weak 5' splice sites followed by a U-rich stretch on their own pre-mRNAs

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Cell proteins TIA-1 and TIAR interact with the 3' stem-loop of the West Nile virus complementary minus-strand RNA and facilitate virus replication

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Nuclear protein TIA-1 regulates COL2A1 alternative splicing and interacts with precursor mRNA and genomic DNA.

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Identification, expression, and tissue distribution of the three rat lysyl hydroxylase isoforms

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Of urchins and men: evolution of an alternative splicing unit in fibroblast growth factor receptor genes

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

TIA-1 is a translational silencer that selectively regulates the expression of TNF-alpha

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

The phenotypic consequences of CFTR mutations

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7 January 2021

SMN2-deletion in childhood-onset spinal muscular atrophy

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Lysyl hydroxylase 2 is a specific telopeptide hydroxylase, while all three isoenzymes hydroxylate collagenous sequences

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Pathogenesis of fibrosis: type 1 collagen and the skin

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Expression profile of proteins involved in scar formation in the healing process of full-thickness excisional wounds in the porcine model

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https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Cloning and characterization of a novel human lysyl hydroxylase isoform highly expressed in pancreas and muscle

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Elevated formation of pyridinoline cross-links by profibrotic cytokines is associated with enhanced lysyl hydroxylase 2b levels

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Identification of PLOD2 as telopeptide lysyl hydroxylase, an important enzyme in fibrosis

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

The type of collagen cross-link determines the reversibility of experimental skin fibrosis

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

RNAi-mediated PTB depletion leads to enhanced exon definition

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Tissue-specific expression and regulation of the alternatively-spliced forms of lysyl hydroxylase 2 (LH2) in human kidney cells and skin fibroblasts.

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Systemic and cell type-specific gene expression patterns in scleroderma skin

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Functional diversity of lysyl hydroxylase 2 in collagen synthesis of human dermal fibroblasts

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Tissue specificity of a new splice form of the human lysyl hydroxylase 2 gene

1 reference

https://api.crossref.org/works/10.1038%2FJID.2008.386

7 January 2021

Identifiers

DOI

10.1038/JID.2008.386

1 reference

20 December 2017

1 reference

20 December 2017