(Q46191238)

English

Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy (English)

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Christian Deschepper

6

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

author name string

Jimmy Matas

1

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Nicholas Tien Sing Young

2

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Céline Bourcier-Lucas

3

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Alexis Ascah

4

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Mariannick Marcil

5

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Yan Burelle

7

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Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

publication date

6 November 2008

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Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Journal of Molecular and Cellular Cardiology

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Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

46

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Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

3

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

420-430

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction

1 reference

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Mitochondrial function in heart muscle from patients with idiopathic dilated cardiomyopathy

1 reference

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Mitochondrial tolerance to stress impaired in failing heart

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Abnormal mitochondrial respiration in failed human myocardium

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NHE-1 inhibition improves impaired mitochondrial permeability transition and respiratory function during postinfarction remodelling in the rat.

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Cyclosporine A attenuates mitochondrial permeability transition and improves mitochondrial respiratory function in cardiomyocytes isolated from dogs with heart failure

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Apoptosis in heart failure: release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy

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Apoptosis in the failing human heart

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Apoptotic pathway activation from mitochondria and death receptors without caspase-3 cleavage in failing human myocardium: fragile balance of myocyte survival?

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Dysfunction of mitochondrial respiratory chain complex I in human failing myocardium is not due to disturbed mitochondrial gene expression

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Apoptosis of ventricular myocytes: a means to an end.

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Myocyte apoptosis in heart failure.

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Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress

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7 January 2021

Mitochondria and cell death. Mechanistic aspects and methodological issues

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https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.10.020

7 January 2021

The mitochondrial permeability transition.

1 reference

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7 January 2021

Recruitment of mitochondrial cyclophilin to the mitochondrial inner membrane under conditions of oxidative stress that enhance the opening of a calcium-sensitive non-specific channel

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.10.020

7 January 2021

Chaotropic agents and increased matrix volume enhance binding of mitochondrial cyclophilin to the inner mitochondrial membrane and sensitize the mitochondrial permeability transition to [Ca2+]

1 reference

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7 January 2021

The mitochondrial permeability transition pore and its role in cell death

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7 January 2021

Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure

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7 January 2021

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

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Simple, rapid, and effective method of producing aortocaval shunts in the rat

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A genetic locus accentuates the effect of volume overload on adverse left ventricular remodeling in male and female rats.

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7 January 2021

Rhodamine 123 as a probe of transmembrane potential in isolated rat-liver mitochondria: spectral and metabolic properties

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7 January 2021

Muscle denervation promotes opening of the permeability transition pore and increases the expression of cyclophilin D

1 reference

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7 January 2021

Superoxide production by the mitochondrial respiratory chain

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7 January 2021

Stress pathways and heart failure

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7 January 2021

Alterations in calcium handling in cardiac hypertrophy and heart failure

1 reference

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7 January 2021

Altered intracellular Ca2+ handling in heart failure

1 reference

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7 January 2021

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.10.020

7 January 2021

Cyclophilins and their possible role in the stress response

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2008.10.020

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Chaperone function of Hsp90-associated proteins

1 reference

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7 January 2021

Molecular chaperone machines: chaperone activities of the cyclophilin Cyp-40 and the steroid aporeceptor-associated protein p23

1 reference

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7 January 2021

Induction of heat shock protein 72 in the failing heart is attenuated after an exposure to heat shock

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7 January 2021

Myocardial heat shock proteins during the development of heart failure

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Changes in Hsp60 level of the failing heart following acute myocardial infarction and the effect of long-term treatment with trandolapril

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7 January 2021

Increased susceptibility of striatal mitochondria to calcium-induced permeability transition

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Role of cyclophilin D in the resistance of brain mitochondria to the permeability transition.

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Involvement of Cyclophilin D in the Activation of A mitochondrial Pore by Ca2+ and Oxidant Stress

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Evidence for the Involvement of a Membrane-Associated Cyclosporin-A-Binding Protein in the Ca2+-Activated Inner Membrane Pore of Heart Mitochondria

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Decreased cardiac mitochondrial NADP+-isocitrate dehydrogenase activity and expression: a marker of oxidative stress in hypertrophy development

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Cardiac mitochondrial NADP+-isocitrate dehydrogenase is inactivated through 4-hydroxynonenal adduct formation: an event that precedes hypertrophy development

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Cyclophilin-D binds strongly to complexes of the voltage-dependent anion channel and the adenine nucleotide translocase to form the permeability transition pore

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Purification and N-terminal sequencing of peptidyl-prolyl cis-trans-isomerase from rat liver mitochondrial matrix reveals the existence of a distinct mitochondrial cyclophilin

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10.1016/J.YJMCC.2008.10.020

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Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

release_bnvjifnhtjfgzf7w64sm4qk4ke

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https://api.fatcat.wiki/v0/release/bnvjifnhtjfgzf7w64sm4qk4ke

24 November 2022

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

20 December 2017

http://europepmc.org/abstract/MED/19094991

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