(Q46421589)

22 December 2017

title

Overweight and obesity associated with a missense polymorphism in fatty acid amide hydrolase (FAAH). (English)

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22 December 2017

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Sipe JC

1

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22 December 2017

Waalen J

series ordinal

2

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22 December 2017

Gerber A

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3

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22 December 2017

Beutler E

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4

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22 December 2017

language of work or name

English

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publication date

1 July 2005

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22 December 2017

International Journal of Obesity

published in

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22 December 2017

volume

29

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22 December 2017

issue

7

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22 December 2017

page(s)

755-759

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22 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.ijo.0802954

exact match

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Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides

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Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase

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Pharmacological activity of fatty acid amides is regulated, but not mediated, by fatty acid amide hydrolase in vivo

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A missense mutation in human fatty acid amide hydrolase associated with problem drug use.

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Obesity wars: molecular progress confronts an expanding epidemic

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The endogenous cannabinoid system affects energy balance via central orexigenic drive and peripheral lipogenesis

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The cannabinoid system: a role in both the homeostatic and hedonic control of eating?

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Reduced cellular expression and activity of the P129T mutant of human fatty acid amide hydrolase: evidence for a link between defects in the endocannabinoid system and problem drug use.

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7 January 2021

The fatty acid amide hydrolase (FAAH)

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7 January 2021

Large-scale screening for HFE mutations: methodology and cost

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Localization and mechanisms of action of cannabinoid receptors at the glutamatergic synapses of the mouse nucleus accumbens.

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Endogenous cannabinoid system as a modulator of food intake

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7 January 2021

Identifiers

DOI

10.1038/SJ.IJO.0802954

1 reference

22 December 2017

PubMed ID

15809662

1 reference

22 December 2017