(Q46507682)

22 December 2017

title

Ischemic injury underlies the pathogenesis of aristolochic acid-induced acute kidney injury (English)

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22 December 2017

author name string

Yu-Jie Wen

series ordinal

1

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22 December 2017

Lei Qu

series ordinal

2

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22 December 2017

Xiao-Mei Li

series ordinal

3

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22 December 2017

language of work or name

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4 June 2008

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22 December 2017

published in

Translational Research

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22 December 2017

volume

152

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22 December 2017

page(s)

38-46

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22 December 2017

issue

1

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22 December 2017

Aristolochic acid and 'Chinese herbs nephropathy': a review of the evidence to date

cites work

1 reference

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Aristolochic acid nephropathy: what we know and what we have to do.

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For patients taking herbal therapy--lessons from aristolochic acid nephropathy

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Rapidly progressive interstitial renal fibrosis in young women: association with slimming regimen including Chinese herbs

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Chinese herbs nephropathy presentation, natural history and fate after transplantation.

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Pathologic aspects of a newly described nephropathy related to the prolonged use of Chinese herbs

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The renin-angiotensin system blockade does not prevent renal interstitial fibrosis induced by aristolochic acids

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Effects of steroids on the progression of renal failure in chronic interstitial renal fibrosis: a pilot study in Chinese herbs nephropathy

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Cytotoxicity of phenanthrenes extracted from Aristolochia contorta in human proximal tubular epithelial cell line.

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Structure activity relationships of aristolochic acid analogues: toxicity in cultured renal epithelial cells

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Role of mitochondrial permeability transition in human renal tubular epithelial cell death induced by aristolochic acid.

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Aristolochic acid impedes endocytosis and induces DNA adducts in proximal tubule cells

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Aristolochic acid induces proximal tubule apoptosis and epithelial to mesenchymal transformation

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Effect of aristolochic acid on intracellular calcium concentration and its links with apoptosis in renal tubular cells

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Possible mechanisms explaining the tendency towards interstitial fibrosis in aristolochic acid-induced acute tubular necrosis.

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Early proximal tubule injury in experimental aristolochic acid nephropathy: functional and histological studies

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Chronic aristolochic acid toxicity in rabbits: a model of Chinese herbs nephropathy?

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Rapidly progressive fibrosing interstitial nephritis associated with Chinese herbal drugs

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Role of peritubular capillary loss and hypoxia in progressive tubulointerstitial fibrosis in a rat model of aristolochic acid nephropathy.

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Stimulation of hypoxia-inducible factor-1 alpha (HIF-1alpha) protein in the adult rat testis following ischemic injury occurs without an increase in HIF-1alpha messenger RNA expression

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Hypoxia-inducible factor modulates tubular cell survival in cisplatin nephrotoxicity

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Post-cyclosporine-mediated hypertension and nephropathy: amelioration by vascular endothelial growth factor

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Hypoxic conditions stimulate the production of angiogenin and vascular endothelial growth factor by human renal proximal tubular epithelial cells in culture

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Effects of hypoxia on growth factor expression in the rat kidney in vivo

7 January 2021

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Induction of VEGF and VEGF receptor gene expression by hypoxia: divergent regulation in vivo and in vitro

7 January 2021

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High glucose blunts vascular endothelial growth factor response to hypoxia via the oxidative stress-regulated hypoxia-inducible factor/hypoxia-responsible element pathway

7 January 2021

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7 January 2021

Identifiers

DOI

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22 December 2017

PubMed ID

18593636

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22 December 2017