(Q46523157)

1 July 2019

title

Deficiency of NG2+ cells contributes to the susceptibility of stroke-prone spontaneously hypertensive rats (English)

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1 July 2019

author

Pei Wang

1

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1 July 2019

Chao-Yu Miao

5

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1 July 2019

author name string

Wei-Wei Tian

2

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1 July 2019

Jie Song

3

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1 July 2019

Yun-Feng Guan

4

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1 July 2019

publication date

1 October 2011

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1 July 2019

CNS Neuroscience & Therapeutics

published in

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1 July 2019

volume

17

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1 July 2019

issue

5

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1 July 2019

page(s)

327-332

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1 July 2019

https://api.crossref.org/works/10.1111%2FJ.1755-5949.2011.00265.X

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NG2 cells generate both oligodendrocytes and gray matter astrocytes.

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Postnatal NG2 proteoglycan-expressing progenitor cells are intrinsically multipotent and generate functional neurons

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Blood pressure variability is more important than blood pressure level in determination of end-organ damage in rats.

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Regional cerebral blood flow, glucose metabolism, protein synthesis, serum protein extravasation, and content of biochemical substrates in stroke-prone spontaneously hypertensive rats.

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Association between short-term exposure to ultrafine particles and hospital admissions for stroke in Copenhagen, Denmark

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Effect of nitro-L-arginine on electrical and mechanical responses to acetylcholine in the superior mesenteric artery from stroke-prone hypertensive rat.

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Altered expression of endothelin and its receptors in the brain of SHR-SP at malignant hypertensive stage.

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Structural alterations of tight junctions are associated with loss of polarity in stroke-prone spontaneously hypertensive rat blood-brain barrier endothelial cells.

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1 reference

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Reduction of cerebral infarction in stroke-prone spontaneously hypertensive rats by statins associated with amelioration of oxidative stress

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Identifiers

DOI

1 reference

1 July 2019

PMCID

6493835

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1 July 2019

PubMed ID

21951366

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1 July 2019