(Q46709852)

24 December 2017

title

Ionizing radiation induces DNA double-strand breaks in bystander primary human fibroblasts (English)

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24 December 2017

author name string

Mykyta V Sokolov

1

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24 December 2017

Lubomir B Smilenov

2

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24 December 2017

Eric J Hall

3

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24 December 2017

Igor G Panyutin

4

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24 December 2017

William M Bonner

5

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24 December 2017

Olga A Sedelnikova

6

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24 December 2017

language of work or name

English

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publication date

1 November 2005

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24 December 2017

published in

Oncogene

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24 December 2017

volume

24

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24 December 2017

issue

49

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24 December 2017

page(s)

7257-7265

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24 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208886

exact match

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cites work

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Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks

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Genomic instability in mice lacking histone H2AX

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H2AX is required for chromatin remodeling and inactivation of sex chromosomes in male mouse meiosis

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Genomic instability and bystander effects induced by high-LET radiation

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Recombinational DNA double-strand breaks in mice precede synapsis

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Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaks

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21 January 2018

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21 January 2018

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21 January 2018

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Histone H2AX: a dosage-dependent suppressor of oncogenic translocations and tumors

21 January 2018

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7 January 2021

The bystander effect in radiation oncogenesis: II. A quantitative model

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7 January 2021

Medium-mediated intercellular communication is involved in bystander responses of X-ray-irradiated normal human fibroblasts

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7 January 2021

Functional hemichannels in astrocytes: a novel mechanism of glutamate release.

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1208886

1 reference

24 December 2017

1 reference

24 December 2017