(Q46712828)

English

High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway

scientific article published in September 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

High glucose-induced human umbilical vein endothelial cell hyperpermeability is dependent on protein kinase C activation and independent of the Ca2+-nitric oxide signalling pathway (English)

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

author name string

Lei Dang

1

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

J Paul Seale

2

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

Xianqin Qu

3

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

publication date

1 September 2005

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

Clinical and Experimental Pharmacology and Physiology

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

32

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

9

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

771-776

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

Protein kinase C phosphorylates caldesmon77 and vimentin and enhances albumin permeability across cultured bovine pulmonary artery endothelial cell monolayers

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Protein kinase signaling in the modulation of microvascular permeability

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Role of Ca2+ signaling in the regulation of endothelial permeability

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Intracellular signalling involved in modulating human endothelial barrier function

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

High glucose concentration causes a rise in [Ca2+]i of cardiac myocytes

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Reduction of high glucose and phorbol-myristate-acetate-induced endothelial cell permeability by protein kinase C inhibitors LY379196 and hypocrellin A.

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Endothelial cell monolayers as a model system to investigate dengue shock syndrome

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Protein kinase C isoenzymes: divergence in signal transduction?

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Regulation of endothelial constitutive nitric oxide synthase by protein kinase C.

1 reference

https://api.crossref.org/works/10.1111%2FJ.1440-1681.2005.04266.X

21 January 2018

Identifiers

10.1111/J.1440-1681.2005.04266.X

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

24 December 2017

http://europepmc.org/abstract/MED/16173935

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