(Q46729188)

24 December 2017

title

Oxidative DNA damage induced by metabolites of chloramphenicol, an antibiotic drug (English)

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24 December 2017

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Shiho Ohnishi

1

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24 December 2017

Mariko Murata

2

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24 December 2017

Naoyuki Ida

3

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24 December 2017

Shinji Oikawa

4

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24 December 2017

Shosuke Kawanishi

5

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24 December 2017

publication date

3 July 2015

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24 December 2017

published in

Free Radical Research

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24 December 2017

volume

49

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24 December 2017

issue

9

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24 December 2017

page(s)

1165-1172

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24 December 2017

Chloramphenicol: a review

cites work

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

21 January 2018

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Chloramphenicol induces abnormal differentiation and inhibits apoptosis in activated T cells

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Synthesis and Antibiotic Properties of Chloramphenicol Reduction Products

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Oxidative DNA damage by vitamin A and its derivative via superoxide generation

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

57 Sequencing end-labeled DNA with base-specific chemical cleavages

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Oxidative damage to cellular and isolated DNA by metabolites of a fungicide ortho-phenylphenol

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Degradation of isolated deoxyribonucleic acid mediated by nitroso-chloramphenicol. Possible role in chloramphenicol-induced aplastic anemia

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

The LEC rat: a useful model for studying liver carcinogenesis related to oxidative stress and inflammation.

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Copper is required for oncogenic BRAF signalling and tumorigenesis

21 January 2018

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Oxidative DNA damage by a metabolite of carcinogenic 1-nitropyrene

21 January 2018

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https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Oxidative DNA damage by a metabolite of carcinogenic and reproductive toxic nitrobenzene in the presence of NADH and Cu(II).

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

The stimulus-secretion coupling of glucose-induced insulin release. XXXV. The links between metabolic and cationic events

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Nonenzymatic reduction of nitro derivative of a heterocyclic amine IQ by NADH and Cu(II) leads to oxidative DNA damage.

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Chloramphenicol: relation of structure to activity and toxicity

21 January 2018

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Reactions of nitrosochloramphenicol in blood

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Aerobic nitroreduction of dehydrochloramphenicol by bone marrow

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Oxidative stress in the regulation of normal and neoplastic hematopoiesis

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Residual Marrow Damage: Possible Explanation for Idiosyncrasy to Chloramphenicol

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Free radical-induced double lesions in DNA.

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Site specificity and mechanism of oxidative DNA damage induced by carcinogenic catechol

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

Oxidatively generated complex DNA damage: tandem and clustered lesions.

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

HO* radicals induce an unexpected high proportion of tandem base lesions refractory to repair by DNA glycosylases.

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F10715762.2015.1050963

10.3109/10715762.2015.1050963

21 January 2018

Identifiers

DOI

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24 December 2017

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24 December 2017