(Q47072128)

5 January 2018

title

Drosophila synaptotagmin I null mutants survive to early adulthood (English)

1 reference

5 January 2018

Synaptotagmin 1 Dmel_CG3139

main subject

Drosophila

0 references

1 reference

GOA release 2020-03-11

author name string

Loewen CA

series ordinal

1

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5 January 2018

Mackler JM

series ordinal

2

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5 January 2018

Reist NE

series ordinal

3

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5 January 2018

publication date

1 September 2001

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5 January 2018

published in

Genesis

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5 January 2018

volume

31

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5 January 2018

issue

1

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5 January 2018

page(s)

30-36

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5 January 2018

Drosophila UNC-13 is essential for synaptic transmission

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1 reference

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GFP-tagged balancer chromosomes for Drosophila melanogaster

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A novel function for the second C2 domain of synaptotagmin. Ca2+-triggered dimerization

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Delineation of the oligomerization, AP-2 binding, and synprint binding region of the C2B domain of synaptotagmin

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Ca2+ regulates the interaction between synaptotagmin and syntaxin 1.

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Molecular mechanisms in synaptic vesicle endocytosis and recycling

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The C2B domain of synaptotagmin is a Ca(2+)-sensing module essential for exocytosis

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Synaptic transmission persists in synaptotagmin mutants of Drosophila

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The effect on synaptic physiology of synaptotagmin mutations in Drosophila

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Synaptotagmin I functions as a calcium regulator of release probability

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Synaptotagmin I: a major Ca2+ sensor for transmitter release at a central synapse

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The C terminus of SNAP25 is essential for Ca(2+)-dependent binding of synaptotagmin to SNARE complexes

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Membrane fusion and exocytosis

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Defective recycling of synaptic vesicles in synaptotagmin mutants of Caenorhabditis elegans.

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Ca(2+)-dependent and -independent activities of neural and non-neural synaptotagmins

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Calcium dependence of neurotransmitter release and rate of spontaneous vesicle fusions are altered in Drosophila synaptotagmin mutants

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Mutational analysis of Drosophila synaptotagmin demonstrates its essential role in Ca(2+)-activated neurotransmitter release.

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Synaptic function is impaired but not eliminated in C. elegans mutants lacking synaptotagmin

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New advances in Drosophila provide opportunities to study gene functions

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Binding of the synaptic vesicle v-SNARE, synaptotagmin, to the plasma membrane t-SNARE, SNAP-25, can explain docked vesicles at neurotoxin-treated synapses

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Genetic and electrophysiological studies of Drosophila syntaxin-1A demonstrate its role in nonneuronal secretion and neurotransmission

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Interaction of the synprint site of N-type Ca2+ channels with the C2B domain of synaptotagmin I.

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A protein assembly-disassembly pathway in vitro that may correspond to sequential steps of synaptic vesicle docking, activation, and fusion

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SNAP receptors implicated in vesicle targeting and fusion

21 January 2018

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Crystal structure of a SNARE complex involved in synaptic exocytosis at 2.4 A resolution

21 January 2018

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The C2B domain of synaptotagmin I is a Ca2+-binding module

21 January 2018

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21 January 2018

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21 January 2018

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7 January 2021

Kinetics of synaptotagmin responses to Ca2+ and assembly with the core SNARE complex onto membranes

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7 January 2021

Calcium-dependent dissociation of synaptotagmin from synaptic SNARE complexes

1 reference

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7 January 2021

Synaptotagmin is an inositol polyphosphate binding protein: isolation and characterization as an Ins 1,3,4,5-P4 binding protein

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7 January 2021

Improved stability of Drosophila larval neuromuscular preparations in haemolymph-like physiological solutions

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7 January 2021

Identifiers

DOI

10.1002/GENE.10002

1 reference

5 January 2018

1 reference

5 January 2018