(Q47260686)

12 January 2018

title

Cardiac contractile dysfunction in J2N-k cardiomyopathic hamsters is associated with impaired SR function and regulation (English)

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12 January 2018

author name string

Andrea P Babick

1

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12 January 2018

Elliott J F Cantor

2

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12 January 2018

John T Babick

3

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12 January 2018

Nobuakira Takeda

4

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12 January 2018

Naranjan S Dhalla

5

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12 January 2018

Thomas Netticadan

6

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12 January 2018

publication date

30 June 2004

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12 January 2018

American Journal of Physiology - Cell Physiology

published in

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12 January 2018

volume

287

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12 January 2018

issue

5

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12 January 2018

page(s)

C1202-8

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12 January 2018

Cardiac excitation-contraction coupling

cites work

1 reference

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Anti-phospholamban and protein kinase A alter the Ca2+ sensitivity and maximum velocity of Ca2+ uptake by the cardiac sarcoplasmic reticulum

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21 January 2018

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Concerted regulation of cardiac sarcoplasmic reticulum calcium transport by cyclic adenosine monophosphate dependent and calcium-calmodulin-dependent phosphorylations

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New molecular insights into heart failure and cardiomyopathy: potential strategies and therapies

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Molecular mechanisms of reduced sarcoplasmic reticulum Ca(2+) uptake in human failing left ventricular myocardium

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Defect of delta-sarcoglycan gene is responsible for development of dilated cardiomyopathy of a novel hamster strain, J2N-k: calcineurin/PP2B activity in the heart of J2N-k hamster

21 January 2018

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Depressed levels of Ca2+-cycling proteins may underlie sarcoplasmic reticulum dysfunction in the diabetic heart

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21 January 2018

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Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, delta-sarcoglycan, in hamster: an animal model of disrupted dystrophin-associated glycoprotein complex

21 January 2018

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21 January 2018

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21 January 2018

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10.1152/AJPCELL.00155.2004

21 January 2018

Identifiers

DOI

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12 January 2018

PubMed ID

15229104

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12 January 2018