(Q47594635)

4 February 2018

title

Atm and c-Abl cooperate in the response to genotoxic stress during nervous system development. (English)

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4 February 2018

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Heather L Miller

1

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4 February 2018

Youngsoo Lee

2

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4 February 2018

Jingfeng Zhao

3

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4 February 2018

Miriam J Chong

4

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4 February 2018

Peter J McKinnon

5

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4 February 2018

publication date

1 October 2003

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4 February 2018

number of pages

8

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Developmental Brain Research

inferred from page(s)

published in

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4 February 2018

volume

145

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4 February 2018

page(s)

31-38

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4 February 2018

issue

1

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4 February 2018

Interaction of c-Abl and p73alpha and their collaboration to induce apoptosis

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Atm-deficient mice: a paradigm of ataxia telangiectasia

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Targeted disruption of the gene encoding DNA ligase IV leads to lethality in embryonic mice

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7 January 2021

Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation

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7 January 2021

Xrcc2 is required for genetic stability, embryonic neurogenesis and viability in mice

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Pleiotropic defects in ataxia-telangiectasia protein-deficient mice

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7 January 2021

p63 and p73 are required for p53-dependent apoptosis in response to DNA damage

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7 January 2021

Constitutive association of BRCA1 and c-Abl and its ATM-dependent disruption after irradiation

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7 January 2021

Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development

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A critical role for DNA end-joining proteins in both lymphogenesis and neurogenesis

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7 January 2021

The pathogenesis of ataxia-telangiectasia. Learning from a Rosetta Stone

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7 January 2021

A role for Abl in Notch signaling.

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7 January 2021

The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damage

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7 January 2021

Synthetic lethality between mutation in Atm and DNA-PK(cs) during murine embryogenesis

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Rhabdomyosarcomas and radiation hypersensitivity in a mouse model of Gorlin syndrome

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7 January 2021

Requirement for Atm in ionizing radiation-induced cell death in the developing central nervous system

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7 January 2021

The many substrates and functions of ATM

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7 January 2021

Functional interaction between DNA-PK and c-Abl in response to DNA damage

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7 January 2021

Essential roles for the Abl and Arg tyrosine kinases in neurulation

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7 January 2021

The genetic defect in ataxia-telangiectasia

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7 January 2021

Defective neurogenesis resulting from DNA ligase IV deficiency requires Atm

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7 January 2021

Ataxia telangiectasia mutated-dependent apoptosis after genotoxic stress in the developing nervous system is determined by cellular differentiation status.

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7 January 2021

ATM dependent apoptosis in the nervous system

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7 January 2021

Early embryonic lethality in PARP-1 Atm double-mutant mice suggests a functional synergy in cell proliferation during development

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7 January 2021

Genetic interactions between ATM and the nonhomologous end-joining factors in genomic stability and development

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7 January 2021

Interaction between ATM protein and c-Abl in response to DNA damage

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7 January 2021

ATM and ATR: networking cellular responses to DNA damage

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7 January 2021

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7 January 2021

Abl: mechanisms of regulation and activation.

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7 January 2021

Neonatal lethality with abnormal neurogenesis in mice deficient in DNA polymerase beta

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7 January 2021

Neonatal lethality and lymphopenia in mice with a homozygous disruption of the c-abl proto-oncogene

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7 January 2021

Cycling, stressed-out and nervous: cellular functions of c-Abl

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7 January 2021

Regulation of cell death by the Abl tyrosine kinase

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7 January 2021

c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency

1 reference

https://api.crossref.org/works/10.1016%2FS0165-3806%2803%2900192-5

7 January 2021

Targeted disruption of ATM leads to growth retardation, chromosomal fragmentation during meiosis, immune defects, and thymic lymphoma

1 reference

https://api.crossref.org/works/10.1016%2FS0165-3806%2803%2900192-5

7 January 2021

p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage

1 reference

https://api.crossref.org/works/10.1016%2FS0165-3806%2803%2900192-5

7 January 2021

10.1016/S0165-3806(03)00192-5

Identifiers

DOI

1 reference

4 February 2018

PubMed ID

14519491

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4 February 2018