(Q47784613)

5 February 2018

title

Retroviral BAX gene transfer fails to sensitize malignant glioma cells to CD95L-induced apoptosis and cancer chemotherapy. (English)

1 reference

5 February 2018

author name string

U Naumann

series ordinal

1

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5 February 2018

M Weller

series ordinal

2

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5 February 2018

language of work or name

English

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publication date

1 August 1998

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5 February 2018

International Journal of Cancer

published in

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5 February 2018

volume

77

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5 February 2018

page(s)

645-648

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5 February 2018

issue

4

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5 February 2018

Overexpression of the death-promoting gene bax-alpha which is downregulated in breast cancer restores sensitivity to different apoptotic stimuli and reduces tumor growth in SCID mice

cites work

1 reference

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The E1B 19K protein blocks apoptosis by interacting with and inhibiting the p53-inducible and death-promoting Bax protein

21 January 2018

1 reference

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Bcl-2 and Bax function independently to regulate cell death

21 January 2018

1 reference

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

21 January 2018

1 reference

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Immunochemotherapy of malignant glioma: synergistic activity of CD95 ligand and chemotherapeutics.

21 January 2018

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Bax homodimerization is not required for Bax to accelerate chemotherapy-induced cell death

21 January 2018

1 reference

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BAX enhances paclitaxel-induced apoptosis through a p53-independent pathway

21 January 2018

1 reference

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Induction of the death-promoting gene bax-alpha sensitizes cultured breast-cancer cells to drug-induced apoptosis

21 January 2018

1 reference

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Protooncogene bcl-2 gene transfer abrogates Fas/APO-1 antibody-mediated apoptosis of human malignant glioma cells and confers resistance to chemotherapeutic drugs and therapeutic irradiation

21 January 2018

1 reference

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Topoisomerase-I inhibitors for human malignant glioma: differential modulation of p53, p21, bax and bcl-2 expression and of CD95-mediated apoptosis by camptothecin and beta-lapachone.

21 January 2018

1 reference

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BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2F%28SICI%291097-0215%2819980812%2977%3A4%3C645%3A%3AAID-IJC27%3E3.0.CO%3B2-3

Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

21 January 2018

1 reference

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Overexpression of bax sensitizes human breast cancer MCF-7 cells to radiation-induced apoptosis

21 January 2018

1 reference

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The Bax alpha:Bcl-2 ratio modulates the response to dexamethasone in leukaemic cells and is highly variable in childhood acute leukaemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2F%28SICI%291097-0215%2819980812%2977%3A4%3C645%3A%3AAID-IJC27%3E3.0.CO%3B2-3

10.1002/(SICI)1097-0215(19980812)77:4<645::AID-IJC27>3.0.CO;2-3

21 January 2018

Identifiers

DOI

1 reference

5 February 2018

PubMed ID

9679771

1 reference

5 February 2018