(Q47849818)

6 February 2018

title

Effects of a dominant interfering mutant of FADD on signal transduction in activated T cells (English)

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6 February 2018

author

Andreas Strasser

4

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6 February 2018

author name string

Newton K

1

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6 February 2018

Kurts C

2

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6 February 2018

Harris AW

3

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6 February 2018

language of work or name

English

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publication date

1 February 2001

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6 February 2018

published in

Current Biology

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6 February 2018

volume

11

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6 February 2018

issue

4

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6 February 2018

page(s)

273-276

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6 February 2018

Death receptors: signaling and modulation

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A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

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p53-dependent impairment of T-cell proliferation in FADD dominant-negative transgenic mice

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A role for FADD in T cell activation and development

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Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

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FADD/MORT1 regulates the pre-TCR checkpoint and can function as a tumour suppressor

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Class I-restricted cross-presentation of exogenous self-antigens leads to deletion of autoreactive CD8(+) T cells

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bcl-2 transgene inhibits T cell death and perturbs thymic self-censorship

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A dominant negative Fas-associated death domain protein mutant inhibits proliferation and leads to impaired calcium mobilization in both T-cells and fibroblasts

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Function and activation of NF-kappa B in the immune system

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The regulation and roles of Rel/NF-kappa B transcription factors during lymphocyte activation

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p53 is required for radiation-induced apoptosis in mouse thymocytes

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T cell receptor antagonist peptides induce positive selection

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Mice lacking the c-rel proto-oncogene exhibit defects in lymphocyte proliferation, humoral immunity, and interleukin-2 expression

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Identifiers

DOI

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6 February 2018

1 reference

6 February 2018